THE NEW SCIENCE OF PHYSICAL HEALTH‪.‬ The New Science of Physical Health

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In today's episode:

My interview with the CEO of a $3 billion corporation​ in 2020 served as the catalyst for creating this groundbreaking health platform. This leader of a multi-billion-dollar business found himself amidst a health crisis that nearly claimed his life. His response to a pivotal question compelled me to save the lives of people in other workplaces. If this industry titan couldn't answer my simple question about his health crisis, then his 4000 employees would likely face similar struggles. It meant their lives would remain under threat from a health catastrophe, like his had seemingly appeared out of nowhere. I couldn't stand idly by and let that happen. So, I combined my 40 year education career with the medical research from over 100 expert PhD's from across the globe to create the first-ever 'Workplace Perfect Health Lesson'.

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In today's episode:

Dr. Peter Attia is a prominent physician, longevity expert, and entrepreneur dedicated to advancing human health and well-being. With a background in mechanical engineering and applied mathematics from Queen's University in Canada, he transitioned to the medical field, earning his medical degree from Stanford University and completing his residency in surgery at the Johns Hopkins Hospital. Dr. Attia's diverse educational background uniquely positions him to bridge the gap between science, technology, and medicine.



I break down a recent interview he did on the Today in America.

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In today's episode:

Raising your CRF score, which reflects your body's ability to utilize oxygen during exercise, triggers several molecular and physiological adaptations that contribute to the formation of new blood vessels (angiogenesis) and the remodeling of existing arteries (arteriogenesis). Here's how these processes occur and contribute to lowering your clinical risk of cardiovascular disease:




Increased Oxygen Demand: When you engage in physical activity, your muscles require more oxygen to meet the increased energy demands. This elevated oxygen demand stimulates the release of signaling molecules such as vascular endothelial growth factor (VEGF) and nitric oxide (NO) within the vascular endothelium.
Stimulation of Angiogenesis: VEGF is a key factor in promoting the growth of new blood vessels (angiogenesis) from pre-existing ones. This process helps to expand the network of blood vessels, improving oxygen delivery to tissues and organs. Enhanced angiogenesis ensures adequate blood supply to active tissues, reducing the risk of ischemia and promoting tissue repair and regeneration.
Promotion of Arteriogenesis: Nitric oxide (NO), produced by endothelial cells in response to increased blood flow and shear stress, plays a crucial role in arteriogenesis.

More inside the episode...

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In today's episode:

Platelets, also known as thrombocytes, are small cell fragments in the blood that play a crucial role in the body's ability to stop bleeding and form blood clots. When there is an injury or damage to blood vessels, platelets become activated and adhere to the site of injury, where they release various substances to initiate the blood clotting process. This is a normal and essential part of the body's response to injury, preventing excessive bleeding.

However, when platelets become overactivated, it can lead to problems, especially in the context of cardiovascular diseases (CVD).

Platelet stickiness refers to the tendency of platelets, small blood cells, to adhere to each other or to the walls of blood vessels upon activation. Normally, platelets circulate in the blood in an inactive state. However, when there's injury or damage to blood vessels, platelets become activated, changing shape and releasing chemical signals that prompt them to stick together (aggregate) and adhere to the damaged vessel walls.



This adhesive property of platelets is crucial in the process of hemostasis, which is the body's mechanism to prevent excessive bleeding. When blood vessels are injured, platelets adhere to the site of injury, forming a plug to seal the damaged area. This initial platelet aggregation is followed by the coagulation cascade, involving the formation of a blood clot to further stem the bleeding.

There are 7 new secrets you will learn about reducing platelet stickiness in this lesson.



While platelet stickiness is essential for preventing excessive bleeding, abnormal or excessive platelet aggregation and adhesion can lead to issues like thrombosis, where blood clots form within blood vessels without an injury or obstruction, potentially causing blockages and leading to conditions such as heart attacks or strokes. Therefore, the regulation of platelet stickiness is crucial for maintaining proper hemostasis and preventing abnormal clotting.

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In today's episode:

Increasing insulin uptake refers to the process by which cells in the body become more responsive and efficient at taking up and utilizing insulin. Insulin is a hormone produced by the pancreas that plays a crucial role in regulating blood sugar levels.

When cells become resistant to insulin or when insulin production is insufficient, it can lead to elevated blood sugar levels, a condition known as insulin resistance or diabetes. Increasing insulin uptake is beneficial in managing blood sugar levels and improving overall metabolic health.

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In today's episode:

Inflammation, from a cardiologist's viewpoint, refers to the body's complex biological response to harmful stimuli, such as pathogens, damaged cells, or irritants, aimed at removing the cause of cell injury, clearing out necrotic cells and tissues damaged from the initial insult, and initiating tissue repair. In the context of cardiology, inflammation often plays a pivotal role in the development and progression of various cardiovascular diseases.



Chronic inflammation, particularly in blood vessels, is implicated in the pathogenesis of atherosclerosis, where the arterial walls become inflamed due to the buildup of fatty deposits and immune cell infiltration. This inflammatory response contributes to the formation of plaques that narrow the arteries, leading to reduced blood flow and potentially causing heart attacks or strokes.



Inflammatory markers like C-reactive protein (CRP) and interleukins are often monitored in cardiovascular assessments to gauge the presence and severity of inflammation, aiding in risk assessment and treatment strategies for cardiovascular diseases. Managing inflammation is increasingly recognized as an important aspect of preventing and treating heart conditions, emphasizing lifestyle modifications, medications, and targeted therapies to mitigate its detrimental effects on the heart and blood vessels.