Simini Boards Cast

Simini Podcasts

  The Simini Boards-Cast is the go-to audio study tool for small animal surgery residents prepping for board exams.  Each episode simplifies high-yield surgical content from trusted sources  — built to help you pass faster and with less stress.  🎧 Audio-based learning for passive study ✂️ Practical relevance for surgical application 🧠 Flashcard-style recaps + board-style questions 📈 Designed with resident + program director input  Whether you're commuting, walking the dog, or post-op, turn that time into surgical mastery.  Subscribe now and get board-ready — fast. 

  1. 15 HRS AGO

    Chapter 101 - Part E: Opening the Airway: Arytenoid Lateralization Surgery

    In this BoardsCast episode, we finish Tobias Chapter 101 — Larynx with the harsh reality of laryngeal paralysis: You can’t fix the nerve; you can only fix the airway. Arytenoid lateralization (“tieback”) is not “restoring function.” It’s a permanent mechanical redesign of the airway gate — forcing the larynx open so the patient can breathe, even though the biologic motor is permanently offline.  You’ll learn: The dominant model: tieback = mechanical redesign (not nerve recovery) Why these dogs crash: inspiration creates a vacuum that pulls flaccid arytenoids inward (the paper-straw effect) What the surgery actually does: a permanent non-absorbable suture from the cricoid → muscular process of the arytenoid to hold one side abducted Why unilateral works: small radius gain = massive resistance drop (Poiseuille’s law) The unavoidable tradeoff: you improve airflow by permanently weakening the seal → aspiration pneumonia is the big complicationThe board answer: unilateral tieback is preferred because bilateral massively increases aspiration risk (near-guaranteed failure rates reported) Key takeaway: when the gate won’t open, you prop it open — and then you manage the aspiration risk forever. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    15 min

  2. 15 HRS AGO

    Chapter 101 - Part D: Diagnosing the Airway Problem: Evaluating the Larynx.

    In this BoardsCast episode, we continue Tobias Chapter 101 — Larynx by deleting the most common diagnostic mistake in upper airway medicine: You cannot diagnose the airway by listening to it. Stridor is an alarm, not a diagnosis. The sound tells you there’s a restriction somewhere, but it cannot tell you what is mechanically failing. This episode rebuilds laryngeal evaluation into the only board-safe framework: Laryngeal disease is diagnosed by watching the gate move. You’ll learn: Why noise is not anatomy (sound ≠ structure) The single question that matters: do the arytenoids abduct on inspiration?Why the exam must be done under light anesthesia with spontaneous breathingThe biggest trap: too deep anesthesia can make a normal larynx look paralyzed (false positive) How paradoxical motion fools people (movement ≠ function; vacuum can “flap” a paralyzed larynx) When to use doxapram to force a clear answer — and why you must be ready to intubate Key takeaway: don’t diagnose from the hallway. Diagnose by visualizing the gate. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    20 min

  3. 15 HRS AGO

    Chapter 101 - Part C: When the Gate Won’t Open: Laryngeal Paralysis

    In this BoardsCast episode, we continue Tobias Chapter 101 — Larynx with one of the most terrifying presentations in small animal practice: The lungs are fine, the airway isn’t blocked, and the dog still can’t breathe. That’s because laryngeal paralysis isn’t a lung disease, it’s a mechanical failure of the airway gate. If you remember one model from this episode, make it this: Laryngeal paralysis = failure of the airway gate to open during inspiration. You’ll learn: Why does normal breathing requires active arytenoid abduction (the doors must be pulled open) The single point of failure: cricoarytenoideus dorsalis (CAD) is the only opener What happens when CAD is offline: arytenoids sit paramedian, leaving a narrow rima glottidis slit Why the crisis accelerates with effort/heat: negative pressure sucks flaccid cartilages inward + Poiseuille’s law (radius ↓ → resistance explodes) The true cause in most cases: recurrent laryngeal nerve dysfunction, often as part of geriatric-onset polyneuropathy The classic patient: older large-breed dogs (Labs/Goldens), males overrepresented The surgical paradox: tieback fixes airflow, but permanently increases aspiration pneumonia risk Key takeaway: this dog isn’t failing to breathe because the lungs can’t work — it’s failing because the gate won’t open. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    17 min

  4. 15 HRS AGO

    Chapter 101 - Part B: The Muscles That Open the Airway: Laryngeal Mechanics

    In this BoardsCast episode, we continue Tobias Chapter 101 — Larynx by dismantling the most dangerous misconception in respiratory mechanics: The airway is not a passive tube, It’s a spring-loaded gate that must be actively pulled open with every breath.  This episode rebuilds the entire “how breathing works” model using one visual you won’t forget: the larynx is a set of heavy double doors — and one muscle is the only motor that opens them. You’ll learn: Why inspiration requires active arytenoid abduction (against vacuum pressure) The key anatomy: arytenoids = doors, rima glottidis = doorwayThe single point of failure: cricoarytenoideus dorsalis (CAD) is the only abductor Why the power cord is vulnerable: recurrent laryngeal nerve = long path, easy to fail Why small narrowing becomes catastrophe: Poiseuille’s law (radius ↓ → resistance explodes) Why heat/panting triggers the spiral: turbulence → edema → tighter airway → panic → collapse The mechanical fix: unilateral tieback = “open enough to breathe, not so open they aspirate” Key takeaway: laryngeal paralysis isn’t lung failure — it’s a gate that never opens. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    21 min

  5. 15 HRS AGO

    Chapter 101 - Part A: The Larynx Is a Gate: The Three Jobs of the Airway Valve

    In this BoardsCast episode, we begin Tobias Chapter 101 — Larynx with the scary clinical paradox: the nose is clear, the trachea is open, but the dog still can’t breathe. That’s because the problem isn’t the “tubing, It’s the gate in the middle — the larynx. This episode rebuilds the mental model the boards want you to have cold: The larynx is a dynamic airway valve with three jobs — and it must switch between them flawlessly: airflow regulation, airway protection, and phonation. You’ll learn: The 3 jobs of the larynx: breathe/protect / voiceThe critical opening: the rima glottidis (the adjustable aperture that determines resistance)Why is inspiration active (muscles must abduct the arytenoids before airflow arrives) Why swallowing requires the “doorframe to move”: larynx + hyoid pulled cranially so the airway can be shieldedThe single point of failure: cricoarytenoideus dorsalis (CAD) is the only openerWhy millimeters matter: Poiseuille’s law (radius ↓ → resistance explodes) The core failure pattern: recurrent laryngeal nerve → CAD failure → paradoxical collapse on inspiration (laryngeal paralysis) Key takeaway: the lungs don’t decide if air gets in — the larynx does. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    23 min

  6. 1D AGO

    Chapter 100 - Part E: The Airway Problem: Overlong Soft Palate & Brachycephalic Syndrome

    In this BoardsCast episode, we finish Tobias Chapter 100 - Palate by reframing brachycephalic airway syndrome with the only honest model: This isn’t “noisy breathing.” It’s an animal forcefully pulling air against a physical wall.  The dominant framework is simple: Brachycephalic syndrome is a packaging error. The skull got shorter — the soft tissue didn’t. And the primary internal culprit is the overlong soft palate, which physically invades the laryngeal opening and starts a cascade of obstruction, negative pressure trauma, and progressive collapse.  You’ll learn: Why the elongated soft palate is the first domino (soft tissue doesn’t “shrink” with a shortened skull) The primary triad: stenotic nares + elongated soft palate + hypoplastic tracheaThe vacuum cascade: obstruction → massive negative pressure → swelling → everted laryngeal saccules → laryngeal collapseThe GI consequence: chronic suction can contribute to hiatal hernia and severe reflux signs The physics that makes it lethal: Poiseuille’s law (small radius loss → huge resistance increase) Why heat kills: they generate heat by breathing work, then can’t cool by panting because airflow is blocked Why timing is everything: early correction prevents irreversible secondary damage Key takeaway: you can’t medicate a bunched-up rug out of a hallway — the fix is mechanical, and earlier is safer. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    19 min

  7. 1D AGO

    Chapter 100 - Part D: Closing the Gap: Principles of Palatal Reconstruction

    In this BoardsCast episode, we continue Tobias Chapter 100 - Palate with the ultimate palatal nightmare: You close the defect perfectly, and the sutures look perfect. Two weeks later, the hole is back. This episode delivers the reframe that fixes most palatal failures: When a palatal repair fails, it’s not a suturing problem. It’s a blood supply + tension problem.  You’ll learn: The 2 non-negotiables: preserve vascular supply and eliminate tensionWhy the major palatine artery is the hard palate lifeline (and where it runs) How tension causes failure two ways: mechanical tearing + microvascular strangulationThe common failure modes: flaps too small, inadequate mobilization, artery injury, and suturing over the voidBoard nuggets: flaps should be ≥1.5× defect, handle with stay sutures (not crushing forceps), and prioritize a two-layer closure with synthetic absorbable monofilament The strategy shift: overlapping flap/pocket concept so the suture line sits on supported tissue, not empty space Key takeaway: palatal reconstruction succeeds only when closure is vascularized and tension-free. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    14 min

  8. 1D AGO

    Chapter 100 - Part C: Clefts and Oronasal Fistulas: When the Barrier Breaks

    In this BoardsCast episode, we continue Tobias Chapter 100 - Palate by reframing palatal disease with the only mental model that matters: This is not a feeding problem.It’s a barrier failure. When the palate fails, the oral cavity and nasal passages stop being separate systems. Food, liquid, and oral bacteria cross into the nose — and the downstream consequence is predictable: chronic rhinitis, nasal reflux, and aspiration risk.  You’ll learn: The separation system: hard palate = wall, soft palate = valveThe 3 ways the barrier breaks: congenital clefts, traumatic clefts, oronasal fistulasWhy congenital clefts are midline defects (failure of palatal shelf fusion ~days 25–28 gestation in dogs) Why neonates fail to thrive: can’t create suction + milk reflux → aspiration pneumoniaWhy traumatic repairs often need delay (especially burns/gunshot): wait for necrosis to “declare” before you rebuild Why dental disease causes the common fistula: maxillary canine roots sit against a paper-thin barrier to the nasal cavity The two surgical rules that decide success: tension-free closure + preserve blood supply (first attempt is your best shot) Key takeaway: when separation fails, every swallow becomes a respiratory event. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    18 min
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About

  The Simini Boards-Cast is the go-to audio study tool for small animal surgery residents prepping for board exams.  Each episode simplifies high-yield surgical content from trusted sources  — built to help you pass faster and with less stress.  🎧 Audio-based learning for passive study ✂️ Practical relevance for surgical application 🧠 Flashcard-style recaps + board-style questions 📈 Designed with resident + program director input  Whether you're commuting, walking the dog, or post-op, turn that time into surgical mastery.  Subscribe now and get board-ready — fast. 

Information

  • Creator
    Simini Podcasts
  • Years Active
    2025 - 2026
  • Episodes
    143
  • Rating
    Clean
  • Copyright
    © 2026 Simini Boards Cast
  • Show Website
    Simini Boards Cast

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