Simini Boards Cast

Simini Podcasts

  The Simini Boards-Cast is the go-to audio study tool for small animal surgery residents prepping for board exams.  Each episode simplifies high-yield surgical content from trusted sources  — built to help you pass faster and with less stress.  🎧 Audio-based learning for passive study ✂️ Practical relevance for surgical application 🧠 Flashcard-style recaps + board-style questions 📈 Designed with resident + program director input  Whether you're commuting, walking the dog, or post-op, turn that time into surgical mastery.  Subscribe now and get board-ready — fast. 

  1. MAR 25

    Chapter 103 - Part E: Oxygen Failure: The 5 Mechanisms That Kill Your Patient

    In this BoardsCast episode, we finish Tobias Chapter 103 — Lungs with the most terrifying clinical moment: You gave oxygen, you increased ventilation, and the patient is still hypoxemic. Now what?  This episode reframes hypoxemia into what it actually is: Not one disease, a classification problem. Because all hypoxemia comes from five mechanisms — and each requires a completely different fix. Miss the category, miss the treatment.  You’ll learn:  The 5 buckets: low FiO₂, hypoventilation, diffusion impairment, V/Q mismatch, shunt Why naming the disease doesn’t solve the immediate oxygen crisis Hypoventilation: CO₂ rises and crowds out oxygen (pump failure)  Diffusion impairment: thickened barrier → O₂ fails first because CO₂ diffuses ~20× faster V/Q mismatch: the most common cause — oxygen helps by overloading the healthy units  Shunt: oxygen-resistant because hemoglobin can’t be “overfilled” — you need recruitment/PEEP The bedside algorithm: check CO₂ → give O₂ → judge response → classify (don’t guess) Key takeaway: You don’t lose patients because oxygen failed. You lose them because you treated the wrong failure. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    17 min

  2. MAR 25

    Chapter 103 - Part D: Oxygen Doesn’t Deliver Itself

    In this BoardsCast episode, we continue Tobias Chapter 103 — Lungs with the trap that kills patients while the monitor looks “fine”: Perfect lungs. perfect PaO₂. perfect SpO₂…and the tissues are still suffocating.  Because oxygen entering the blood doesn’t matter if it isn’t being carried and moved. This episode installs the single governing framework for oxygen delivery: DO₂ = Cardiac Output × CaO₂ And then we break down the part most people misunderstand: CaO₂ is almost entirely hemoglobin, not dissolved oxygen. PaO₂ is a “warehouse” number — but hemoglobin is the truck fleet, and cardiac output is the highway.  You’ll learn:  Why no hemoglobin = no delivery (period)  CaO₂ math in plain terms: ~98% carried on hemoglobin, ~1–2% dissolved (PaO₂ barely moves the needle)  Why does doubling PaO₂ barely increase total oxygen content (you doubled the “rounding error”)  4 classic delivery failures the boards love: Severe anemia (missing trucks) Carbon monoxide poisoning (trucks hijacked + won’t unload; pulse ox can lie) Shock/low CO (highway jam) Left shift (doors won’t open: alkalosis/hypothermia/low CO₂)  The bedside pivot: if PaO₂ is normal but the patient is hypoxic, stop chasing lungs and start chasing Hb and flowKey takeaway: oxygen doesn’t move itself — hemoglobin moves it, and the heart delivers it. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    20 min

  3. MAR 25

    Chapter 103 - Part C: Air Isn’t Enough: It Has to Meet Blood

    In this BoardsCast episode, we continue Tobias Chapter 103 — Lungs with the ultimate bedside frustration: You ventilated perfectly; the chest rises, the waveform looks great, and the oxygen is still low.  This episode installs the mental model that fixes the mistake everyone makes in hypoxemia: Oxygenation is not an airflow problem — it’s a matching problem. Air alone is useless without blood. Blood alone is useless without air. Oxygen only happens when they meet at the same microscopic interface.  You’ll learn:  The “speed dating” model: ventilation (V) has to meet perfusion (Q) Why diffusion is distance-limited (air can’t “wander” to find blood)  The two extreme failures: Dead space = ventilation without perfusion (high V/Q) Shunt = perfusion without ventilation (V/Q = 0)  Why shunt crushes oxygen faster than dead space (healthy alveoli can’t “overfill” hemoglobin)  The bedside rule that never lies: severe hypoxemia + poor response to 100% O₂ = true shunt The fix depends on what’s broken:  Dead space → restore perfusion / stop crushing capillaries  Shunt → reopen/fill alveoli (PEEP, drain pleural space, clear fluid) Key takeaway: Air isn’t enough. It has to meet blood. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    21 min

  4. MAR 25

    Chapter 103 - Part B: The Oxygen Problem Isn’t Airflow: Diffusion Mechanics

    In this BoardsCast episode, we continue Tobias Chapter 103 — Lungs by confronting the ICU trap everyone falls into: You crank FiO₂ to 100%, you increase ventilation, and the patient is still hypoxemic. Because you’re trying to treat an oxygen-transfer problem with an airflow solution.  This episode rebuilds hypoxemia into a board-clean classification framework: All hypoxemia comes from 5 mechanisms — and each one needs a different fix. You’ll learn:  The 5 causes of hypoxemia (not 50): low FiO₂, hypoventilation, diffusion impairment, V/Q mismatch, shunt  Why panting can still be hypoventilation (effort ≠ alveolar ventilation; dead space trap)  Fick’s Law in plain language: surface area, gradient, thickness, gas properties Why O₂ fails before CO₂: CO₂ diffuses ~20× faster (sprinter vs slow walker)  The bedside decision tools: CO₂ first (hypoventilation = CO₂ high), then A–a gradient to prove gas-exchange failure  The defining clue: hypoxemia resistant to 100% O₂ = true shunt until proven otherwise Key takeaway: Ventilation treats CO₂. Diffusion determines O₂. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    21 min

  5. MAR 25

    Chapter 103 - Part A: The Lung Is Not a Pump - Ventilation Mechanics: Why Air Moves Without Being Pulled In

    In this BoardsCast episode, we begin Tobias Chapter 103 — Lungs by destroying the most persistent illusion in anatomy: The lungs do not pull air in; there is no suction. The lung is not a pump.  Air moves for one reason only: pressure gradients. And the body creates pressure gradients by changing volume — not by “pulling.” This episode rebuilds breathing from the physics up, using the dominant rule that explains every respiratory disease that follows.  You’ll learn:  The hard rule: Flow = pressure gradient ÷ resistance The dominant model: balloon in a box (lungs in the thorax)  Why inspiration is active: diaphragm expands the “box” → alveolar pressure drops → air flows in  Why normal expiration is passive: elastic recoil shrinks the “box” → pressure rises → air flows out  The 3 forces ventilation must overcome: elastance, surface tension, airway resistance  Compliance vs elastance: stiff lungs = hard to inflate; overly compliant lungs = air trapping (emphysema)  Why small airway narrowing is deadly: radius to the 4th power (½ radius = 16× resistance)  The clinical framework: every breathing problem is a failure of pressure, compliance, or resistanceKey takeaway: You don’t suck air in. You create the conditions that make air move. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    19 min

  6. MAR 17

    Chapter 102 - Part E: Fixing the Airway: Stents, Surgery, and Tradeoffs

    In this BoardsCast episode, we finish Tobias Chapter 102 — Trachea and Bronchi with the most important airway truth: You open the airway… and you create a new problem. This episode reframes “fixing” tracheal collapse as what it actually is: a controlled mechanical compromise. You are not restoring normal biology — you are stabilizing a failing system, then managing the new failure modes you introduced.  You’ll learn: The required mental model: airway treatment stabilizes—it doesn’t restore Why hardware resets the clock: instability traded for new failure modesIntraluminal nitinol stents: instant lumen, chronic irritation → cough → fatigue/fracture Extraluminal polypropylene rings: durable support, but high-risk territory (RLN injury + ischemia) The decision matrix: location (cervical vs intrathoracic), patient risk, owner tolerance The 3 predictable clinician errors: expecting a cure, ignoring long-term complications, choosing based on comfort vs anatomy Key takeaway: You didn’t fix the airway. You changed the problem. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    19 min

  7. MAR 17

    Chapter 102 - Part D: Diagnosing a Moving Target: Imaging the Airway

    In this BoardsCast episode, we continue Tobias Chapter 102 — Trachea and Bronchi with the most frustrating clinic moment: The dog is coughing, struggling, even collapsing… And the chest radiographs look normal.  This episode installs the core framework that fixes that mismatch: Airway disease is dynamic — diagnosis fails when imaging is static. A radiograph is a split-second snapshot of a moving system. If you capture the wrong millisecond (wrong phase of the breath), you can miss severe collapse and falsely reassure yourself the airway is “fine.”  You’ll learn: Why the trachea is a pressure-dependent structure (not rigid PVC) Why static X-rays miss phase-dependent collapse (timing lottery) The segment/phase rule:The cervical trachea collapses on inspirationIntrathoracic trachea collapses on expirationWhy fluoroscopy wins: it’s a video, not a photo—watch through breathing + induced cough Why tracheobronchoscopy matters: direct visualization + grading + mucosal health The grading logic (I–IV): dorsal membrane laxity → cartilage flattening → severe inflammation/ciliary failure → complete collapse Key takeaway: don’t ask “what does the airway look like?” — ask “when does the airway fail, and how do I capture it?” 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    17 min

  8. MAR 17

    Chapter 102 - Part C: Tracheal Collapse: The Disease You Can Hear

    In this BoardsCast episode, we continue Tobias Chapter 102 — Trachea and Bronchi with the clinical sound you recognize before you even see the dog: that dry, honking “goose” cough. It sounds like irritation. It sounds like an infection, but it’s neither. Its structure is failing under pressure. Tracheal collapse isn’t a fixed obstruction — it’s a dynamic mechanical failure where normal breathing pressures expose a weakened airway, like a cheap vacuum hose that looks fine until suction starts.  You’ll learn: The core model: structural weakness + normal pressure changes = collapseNormal trachea anatomy: 35–46 C-shaped rings + dorsal trachealis membrane What fails in collapse: cartilage loses strength (GAG loss → dehydration) + dorsal membrane becomes lax and sags Why it’s so “on/off”: the same airway behaves differently at rest vs excitement/exercise The phase/location rule:Cervical (neck) collapse = inspirationIntrathoracic (chest) collapse = expirationThe feedback loop that worsens everything: collapse → irritation → cough → pressure spikes → more collapse Why resting radiographs can miss it (dynamic problem) and why fluoroscopy/tracheoscopy are gold standards Key takeaway: you’re not hearing infection — you’re hearing mechanics failing under pressure. 🎁 Simini Bonus Claim your free sample of Simini Protect Lavage (just cover shipping): https://www.simini.com/evaluation-kit Listen On: Spotify | Apple Podcasts | Amazon Music

    13 min
See All (153)

Ratings & Reviews

5
out of 5
2 Ratings

About

  The Simini Boards-Cast is the go-to audio study tool for small animal surgery residents prepping for board exams.  Each episode simplifies high-yield surgical content from trusted sources  — built to help you pass faster and with less stress.  🎧 Audio-based learning for passive study ✂️ Practical relevance for surgical application 🧠 Flashcard-style recaps + board-style questions 📈 Designed with resident + program director input  Whether you're commuting, walking the dog, or post-op, turn that time into surgical mastery.  Subscribe now and get board-ready — fast. 

Information

  • Creator
    Simini Podcasts
  • Years Active
    2025 - 2026
  • Episodes
    153
  • Rating
    Clean
  • Copyright
    © 2026 Simini Boards Cast
  • Show Website
    Simini Boards Cast

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