Fork U with Dr. Terry Simpson

Terry Simpson
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Fork U(niversity) Not everything you put in your mouth is good for you. There’s a lot of medical information thrown around out there. How are you to know what information you can trust, and what’s just plain old quackery? You can’t rely on your own “google fu”. You can’t count on quality medical advice from Facebook. You need a doctor in your corner. On each episode of Your Doctor’s Orders, Dr. Terry Simpson will cut through the clutter and noise that always seems to follow the latest medical news. He has the unique perspective of a surgeon who has spent years doing molecular virology research and as a skeptic with academic credentials. He’ll help you develop the critical thinking skills so you can recognize evidence-based medicine, busting myths along the way. The most common medical myths are often disguised as seemingly harmless “food as medicine”. By offering their own brand of medicine via foods, These hucksters are trying to practice medicine without a license. And though they’ll claim “nutrition is not taught in medical schools”, it turns out that’s a myth too. In fact, there’s an entire medical subspecialty called Culinary Medicine, and Dr. Simpson is certified as a Culinary Medicine Specialist. Where today's nutritional advice is the realm of hucksters, Dr. Simpson is taking it back to the realm of science.

  1. Willpower Is B.S.: A Surgeon on Zepbound

    3 DAYS AGO

    Willpower Is B.S.: A Surgeon on Zepbound

    Willpower Is B.S.: Food Noise, Healthspan, and What Actually Changed My LifeFor decades, I started every New Year the same way. In January, I promised myself this would be the year. By February, I tried harder. Every spring, I adjusted the plan. And by summer or fall, the weight crept back. That cycle repeated not because I lacked knowledge, discipline, or effort. Instead, it repeated because I misunderstood biology — at least when it came to myself. This year is different. For the first time since Ronald Reagan was first elected, weight loss is not at the top of my New Year’s resolution list. Not because I stopped caring, but because I lost 45 pounds with the help of Zepbound over the last year. More importantly, however, I learned something that reshaped how I think about obesity, healthspan, and shame. Before anything else, let me be clear: this is not medical advice. This is a story. Anecdotes are not evidence, even when the anecdote is from a physician. Nevertheless, stories help us understand science when data alone fails to move us. And this story matters. I Had Willpower. That Wasn’t the Problem.For years, people told me — and millions of others — the same thing: move more and eat less. At first glance, that advice sounds logical. After all, calories matter. Energy balance matters. However, reality is more complicated. To begin with, I am a surgeon. Surgical training requires extraordinary willpower. Moreover, I’ve logged food meticulously, cooked Mediterranean-style meals, exercised consistently, and followed every evidence-based recommendation I’ve ever given patients. Meanwhile, Oprah has willpower. Olympic athletes have willpower. Yet obesity persists. Sure, willpower works briefly. In fact, go on a liquid protein diet, and the weight will fall off quickly. Unfortunately, the food noise remains. Eventually, biology wins. Always. In the same way you cannot positive-think your way out of hypertension, cholesterol, diabetes, cancer, or heart disease, you cannot willpower your way out of obesity. Obesity is a disease. It is not a moral failure. Ironically, I knew this intellectually. Nevertheless, I failed to apply it to myself. We have a name for that: cognitive dissonance. Food Noise Was the Missing ConceptThe real turning point did not come from reading another study. Instead, it came from listening to people I trusted. One colleague quietly lost weight on a GLP-1. Another friend told me something more striking: the food noise stopped. Alcohol lost its appeal. Smoking no longer called. That phrase — food noise — suddenly explained decades of struggle. To illustrate, think of sleeping near Lake Shore Drive in Chicago. At first, traffic noise dominates your awareness. Eventually, it fades into the background. Only when you leave the city do you realize how loud it was. Food noise works the same way. When GLP-1 therapy quieted that background signal, eating slowed naturally. Meals ended without effort. Desire changed without rules. Biology shifted. Notably, calories did not lower my stress. Calories did not improve my sleep. Calories did not stop snoring. Biology did. The Unexpected Early BenefitsInterestingly, weight loss was not the first change I noticed. Sleep improved almost immediately. Stress dropped dramatically. Commutes that once registered hours of physiologic stress now barely registered minutes. Appetite normalized. Eating slowed. These changes matter because sleep and stress directly affect inflammation, metabolic health, appetite signaling, and long-term disease risk. In other words, healthspan improved before the scale reflected anything meaningful. That...

    18 min
  2. Is Whoop Predicting My Death?

    25/12/2025

    Is Whoop Predicting My Death?

    Is Your Watch Predicting Your Death?What Biologic Age Really Means — and What It Doesn’tMy Whoop tells me I’m eight years older than I actually am. Naturally, that raises a question. Does that mean I’m going to die eight years sooner? Is my watch quietly chiseling a new date onto my tombstone? Fortunately, the answer is no. Still, confusion around biologic age has exploded. Wearables promise insight. Apps offer scores. Some even whisper about your future health, as if destiny lives on your wrist. So let’s slow this down and talk about what biologic age really is — and why it matters far less than you think. The Two Numbers and the DashEvery tombstone has two numbers. One marks when you were born. The other marks when you died. However, the most important part isn’t either number. It’s the dash in between. That dash represents your life. It reflects your health, mobility, independence, and curiosity. When we talk about longevity, we shouldn’t obsess over the second number. Instead, we should focus on making those two numbers far apart — and keeping the dash strong for as long as possible. That’s healthspan. Why Biologic Age Sounds Scarier Than It IsBiologic age is not a prophecy. It isn’t a death clock. It doesn’t predict how long you’ll live. Instead, biologic age is a model. It estimates how your body is functioning right now based on things like: resting heart rateheart-rate variabilitysleep duration and consistencyactivity and recovery patternssometimes weight or blood pressure Different devices use different inputs. As a result, they often give different answers. In other words, biologic age reflects recent stress and behavior, not your destiny. Think of it as feedback — not fate. Why Your Watch Isn’t Measuring “Real” AgingEarlier in the Fork U longevity series, we talked about telomeres. Those shorten slowly over decades, one cell division at a time. Your wearable isn’t tracking that. Instead, devices like Whoop measure physiology, not DNA. They detect how hard you’ve been living lately, not how much time you have left. A bad week of sleep, travel, stress, or alcohol can push your biologic age higher. A calm, consistent routine can bring it back down. That’s not aging. That’s load management. A Simple Experiment That Tells the Whole StoryHere’s a trick I tried. I told Whoop I was younger than I actually am. Guess what happened? Suddenly, my biological age dropped below my real age. That alone tells you everything. Whoop isn’t predicting where you’re going. It’s comparing how you’re doing relative to the age you told it you are. Once again, that’s feedback — not destiny. Why I Prefer WithingsI use multiple devices because, frankly, I’m a nerd. However, I tend to prefer Withings for one simple reason. They don’t try to scare you. Instead of telling you how old you “really” are, Withings focuses on things that actually improve your life today: blood pressure trendsbody weight and compositionheart rhythmsleep durationlong-term consistency More importantly, they ask better questions. Are you sleeping better? Is your blood pressure improving? Are your habits trending in the right direction? That’s medicine. Not numerology. And no — Withings didn’t pay me to say that. The Biggest Mistake People...

    10 min
  3. GLP-1 Drugs, the Mediterranean Diet, and the Science of Living Longer

    18/12/2025

    GLP-1 Drugs, the Mediterranean Diet, and the Science of Living Longer

    GLP-1 Drugs, the Mediterranean Diet, and the Science of Living LongerFor years, anti-aging has been hijacked by supplements, hacks, and promises that never hold up. Meanwhile, real science has quietly moved forward. Today, the most compelling anti-aging story does not come from a powder, a cold plunge, or a fasting app. Instead, it comes from metabolism. A class of medications called GLP-1 receptor agonists started as diabetes drugs. Over time, clinicians discovered something bigger. These medicines now play a major role in obesity treatment, and they produce effects that reach far beyond the scale. Because obesity shortens lifespan and damages nearly every organ system, it makes sense that drugs that treat obesity could also improve healthspan—the years you live with strength, clarity, and independence. However, weight loss alone does not explain what researchers are seeing. These drugs reduce inflammation, protect the heart, lower biological stress, and may even delay cognitive decline. Importantly, many of these effects occur independent of weight loss. That fact has forced scientists to ask a serious question: could GLP-1 drugs represent a new class of anti-aging medicine? Even longevity-focused clinicians, such as Peter Attia, have publicly discussed using GLP-1 drugs at lower doses in select patients—not for weight loss, but for metabolic health and long-term disease prevention. Why Metabolism Matters for AgingAging is not just about time. Instead, it reflects how well your body regulates key systems over decades. Blood sugar control, inflammation, oxidative stress, and cellular repair all shape how fast—or how slowly—you age. GLP-1 receptor agonists influence all these pathways. Originally designed to mimic a gut hormone that signals fullness, these drugs turned out to do much more. Research shows they lower systemic inflammation, improve mitochondrial function, and reduce oxidative stress. As a result, organs function better for longer. In simple terms, when metabolism runs smoothly, cells behave younger. Retatrutide and the Next Generation of GLP-1 DrugsNewer drugs have taken this concept even further. Retatrutide, a triple-agonist medication, targets three hormonal pathways simultaneously: GLP-1, GIP, and glucagon. In Phase 3 trials, participants lost nearly 29% of their body weight, or more than 70 pounds on average. Yet weight loss only tells part of the story. Retatrutide also lowered inflammation, improved blood pressure, improved lipid profiles, and reduced joint pain. Each hormone plays a role. GLP-1 reduces appetite and inflammation. GIP improves insulin sensitivity and nutrient handling. Glucagon increases energy expenditure and fat oxidation. Together, these pathways keep metabolism active, not slowing down during weight loss. That combination does more than shrink waistlines. It restores metabolic flexibility, which declines with age. Inflammation: The Engine of AgingFor decades, scientists blamed aging on simple wear and tear. Modern research tells a different story. Chronic, low-grade inflammation—often called inflammaging—drives many diseases of aging. Heart disease, stroke, arthritis, fatty liver disease, and cognitive decline all share this inflammatory background. In clinical trials, GLP-1 drugs reduced markers such as C-reactive protein, triglycerides, and blood pressure. These changes signal reduced biological aging risk, not just better lab numbers. When inflammation falls, fewer senescent cells accumulate. Blood vessels stay healthier. Organs function longer. Heart Disease and...

    12 min
  4. Alcohol Cuts Healthspan

    11/12/2025

    Alcohol Cuts Healthspan

    The Holiday Party That Turned DeadlyIt started at a holiday party. Laughter, champagne, a toast — then a collapse. A fifty-two-year-old, active and healthy, suddenly lost consciousness. Paramedics did CPR and shocked her heart twice. She survived — barely. Doctors called it Holiday Heart Syndrome: an alcohol-triggered arrhythmia that can kill.​ What Is Holiday Heart?Holiday Heart arises after binge or even moderate drinking, especially around celebrations. Alcohol irritates heart cells, disrupts electrolytes, and scrambles electrical signals, which can trigger atrial fibrillation — an erratic rhythm that raises the risk of clots, stroke, and sudden death. Even a single heavy night can set it off, and repeated use amplifies inflammation and structural damage long after the hangover fades.​ Alcohol and Your HeartFor years, the “French paradox” suggested red wine protects the heart, but newer evidence points instead to lifestyle patterns rather than wine itself. Ethanol and its metabolite acetaldehyde directly injure heart muscle, disturb calcium handling, damage mitochondria, and can lead to Alcoholic Cardiomyopathy — an enlarged, weakened heart. Harm shows up even in relatively low intake, and improvement typically requires reducing or stopping alcohol.​ Alcohol and CancerAlcohol is a proven carcinogen that promotes DNA damage, inflammation, oxidative stress, and hormonal shifts that favor tumor growth. At least seven cancers — including those of the mouth, throat, larynx, esophagus, liver, colon, and breast — are directly linked to alcohol, with risk beginning above zero and rising with each additional drink. Even up to one drink a day meaningfully increases breast cancer risk, and the combined use of alcohol and tobacco multiplies risk even further.​ Blue Zones, Not Blue WineYou’ve probably heard this one: People in Sardinia or Ikaria drink wine every night and live to 100. What’s missing is the math. They sip 3 to 4 ounces — not a glass, not a typical American glass, but a tasting. The flight of wine. Their rustic wines are 10–11 percent alcohol, not the 16 percent bombs from Sonoma. And they don’t live long because of the wine. They live long because of everything else: walking hills, eating beans, taking naps, sleeping well, and belonging to a community. Their wine is cultural, not clinical. If you want their healthspan, copy their diet, movement, and purpose — not the nightly pour. Weight, Metabolism, and AgingAlcohol hijacks metabolism by forcing the liver to prioritize ethanol breakdown, pushing fat and sugar processing aside. Drinks can add substantial hidden calories, promote fatty liver, and stall fat loss, even when the rest of a diet looks reasonable.​ Why “Detox” Fixes FailPopular “alcohol detox” supplements promise faster clearance or hangover prevention, but research points to ethanol itself and the inflammatory response as the main drivers of symptoms. Blocking acetaldehyde alone does not prevent mitochondrial damage, immune activation, or the residual effects that follow a night of heavy drinking.​ The Longevity HypocrisyModern wellness culture often warns about “toxins” while normalizing regular drinking, even framing certain spirits or wines as health tools. Yet, when viewed through a longevity lens, alcohol stands out as one of the most potent, fully optional biological stressors in the modern lifestyle.​ When You StopOnce drinking stops or drops sharply, the body begins to repair: blood

    12 min
  5. Muscle, Mitochondria, and Healthspan

    04/12/2025

    Muscle, Mitochondria, and Healthspan

    Muscle is Medicine: Why Lifting Weights is Your Best Longevity InvestmentClearly, your body changes as you age. I learned this lesson years ago when my son was three years old. We started him skiing, and he loved every minute of it. When he fell, he tumbled onto his behind, jumped right back up, and skied down the hill like nothing had happened. He was pure rubber and resilience. However, I was 53 years his senior that year. I did an inadvertent 360-degree twirl on the slopes myself. His mother saw me and immediately asked if I had broken my wrist, wondering when I could return to surgery. The difference between a flexible young body and an older body is critical. Consequently, I retired from skiing that season and now enjoy the lodge, where I write and make them great dinners. Indeed, your older body desperately needs work to stay flexible, strong, and balanced as time goes on. I have seen too many independent seniors lose everything after a simple fall in their own home. They go from living on their own to spending their last days in a care center, sometimes never leaving bed. This outcome is not healthspan. Instead, you want a fall to be like my son’s—just on your butt and back up. Sadly, too many fall and cannot get up. This isn't a commercial for a safety pendant, but a sincere plea for you to start working your muscles. Section 1: The Enemy is Muscle Loss (Sarcopenia)Specifically, we talk frequently about heart health and clear arteries in longevity. Those things are unquestionably crucial. Nevertheless, the biggest threat to functional independence as we age is a condition called sarcopenia. This is the medical term for age-related muscle loss. Unfortunately, we start losing about 3 to 8 percent of our muscle mass every decade after age 30. That loss accelerates quickly once you hit 70. This problem is not just about looking less toned; fundamentally, it is about losing the ability to stand up from a chair, carry groceries, or, most importantly, catch yourself when you trip. The falls that result are often catastrophic. Section 2: Big Things Help Small Things—The Cellular ConnectionAmazingly, resistance training is effective at the microscopic level, too. We have talked extensively about the tiny, complex mechanisms of the cell, but here is the key takeaway: small things benefit from big things. In fact, increasing muscle mass through training has direct, positive effects on two major microscopic drivers of aging: mitochondrial function and telomere health. To elaborate, when you challenge your muscles, you signal your cells to create more energy. This signal forces your mitochondria—the cellular powerhouses—to become both more numerous and more efficient. Better mitochondrial function equals more energy and less cellular stress. Moreover, studies show that resistance training actually increases the activity of the enzyme telomerase in some cells. Telomerase helps maintain the protective caps on your DNA called telomeres. Therefore, you don’t need to buy fancy, expensive supplements like NAD or telomere boosters. Picking up a dumbbell costs less money but yields more results. You gain muscular strength, better metabolism, stronger bones, and the cellular benefits all at once. Section 3: Muscle is Your Metabolic PowerhouseLet's consider how muscle mass influences your diet. Your muscle is actually your body’s largest organ for glucose disposal. Think of it like this: when you eat, your body releases glucose (sugar) into your bloodstream....

    13 min
  6. Telomeres and Time: Rewind Aging

    27/11/2025

    Telomeres and Time: Rewind Aging

    🧬 Telomeres and Time: Can We Really Rewind Aging?The Lowest Hemoglobin I’ve Ever SeenThe lowest hemoglobin I’ve ever seen belonged to a young woman who was still standing. Her blood count was one-fourth of normal. She was pale, short of breath, and strong enough to walk into the clinic. Doctors soon learned her bone marrow had stopped making new blood cells. The diagnosis was aplastic anemia — a true telomere disease. She survived thanks to her fitness, modern science, and a bone marrow transplant from a generous donor in Germany. Two years later, she’s in law school, healthy, and full of life. What Are Telomeres?Each cell in your body carries chromosomes — long strands of DNA. At the ends of those chromosomes sit telomeres, tiny caps that keep the DNA from unraveling, like plastic tips on shoelaces. Every time a cell divides, its telomeres shorten a little. When they get too short, the cell can no longer divide. Scientists call that stage cellular senescence — cellular retirement. In 2009, researchers Elizabeth Blackburn and Carol Greider won the Nobel Prize for discovering telomerase, an enzyme that can rebuild telomeres. Their discovery sparked dreams of reversing aging. But there’s a catch: cancer cells also use telomerase to live forever. Turning that enzyme on everywhere might turn back time — or turn on tumors. Why Everyone Talks About TelomeresTelomeres became the poster child for longevity marketing. Social media ads promise to “measure your biological age.” Supplement companies claim to “lengthen your telomeres” for hundreds of dollars a bottle. The problem? Telomere tests vary between labs. Results can change by 20 percent depending on the method. They show trends, not destiny. What’s Being StudiedReal scientists are studying how telomeres behave under different conditions. Danazol — a synthetic sex hormone that slows telomere loss in people with inherited marrow failure. It works but brings side effects, so it’s not an anti-aging trick.Henagliflozin — a diabetes drug that increased telomere length in one small study. Whether that helps humans live longer is still unknown.Aripiprazole — an antipsychotic that repaired telomeres in cells after oxidative stress. That’s a Petri dish result, not a prescription for youth. These drugs show that we can nudge biology, but they’re for disease, not for vanity. Vitamins and Compounds That Might HelpNutrients influence telomere health, too. Vitamin D supports telomerase. Long-term studies show it slows telomere shortening.Vitamins C and E help reduce chemical stress that wears telomeres down.Gamma-tocotrienol, a form of vitamin E, may reverse telomere loss — so far only in lab work.TA-65, from the Astragalus plant, may activate telomerase but carries risk. Turning on telomerase could also fuel cancer.Telomir 1 is experimental and not available outside research. None of these is proven to extend life. They’re promising ingredients, not miracles in a capsule. What Lifestyle Still Beats EverythingLifestyle matters more than any supplement. A large study at UCSF showed that people who ate a Mediterranean diet, exercised, and managed stress boosted telomerase activity within months. No powder required. Telomeres respond to care. They’re markers of how you live, not the cause of how long you live. Longer telomeres don’t guarantee longer life — they reflect how your body has handled time, inflammation, and stress. What Scientists Agree

    10 min
  7. Mitochondria Matter: The Story of Aging

    20/11/2025

    Mitochondria Matter: The Story of Aging

    The Mitochondria Problem: Why These Tiny Powerhouses Shape How We AgeMany people suddenly talk about mitochondria. You hear them in political speeches, on podcasts, and across social media. RFK Jr said he can “see” kids with weak mitochondria just by watching them walk through an airport. Others claim special diets or powders can “fix” aging by supercharging these organelles. However, most of that chatter misses the actual science. This post breaks down what mitochondria do, why they matter for aging, and how you can keep them healthy. No hype. No detox teas. Just biology you can use. What Are Mitochondria?Every cell in your body contains tiny structures called mitochondria. They act like miniature cells living inside your larger cells. Each mitochondrion even has its own DNA. Mitochondria divide independently from your regular cells. They manage your energy, converting glucose to ATP Finally, mitochondria keep your organs working. You inherit all your mitochondria from your mother, which is why scientists use mitochondrial DNA to trace ancestry. How Did We Get Mitochondria? (A Very Old Story)About 1.5 billion years ago, a simple cell swallowed a bacterium and refused to digest it. Instead, they formed a partnership. The bacterium supplied energy. The host cell provided safety. That partnership became the mitochondrion. Every person alive today runs on that ancient deal. What Do Mitochondria Do All Day? Mitochondria take glucose from your food and convert it into ATP — the energy your body uses to move, think, heal, and grow. This process runs every second of your life. You cannot swallow ATP and get more energy. ATP supplements don’t work. Only your mitochondria make the usable fuel your body needs. Why Young Mitochondria Work So WellYoung mitochondria act like teenagers. They run fast, bounce back quickly, and handle stress with ease. Cells constantly recycle old mitochondria through a process called mitophagy. This system works beautifully in childhood. Fresh mitochondria power: strong musclessharp thinkingfast recoveryhealthy metabolism When mitophagy runs smoothly, you feel energetic and resilient. What Happens When Mitochondria AgeAging slows everything down. Mitochondria begin to leak more “exhaust,” build up mutations, and lose efficiency. Damaged ones don’t get removed as well, because mitophagy weakens with age. Unfortunately, mitochondria do something worse than slow down: They fuse with healthy mitochondria. Imagine pouring spoiled milk into a fresh gallon. The whole jug goes bad. Aging mitochondria do the same thing inside your cells. They spread dysfunction to the healthy ones. How Aging Mitochondria Cause TroubleAs mitochondria fail, they change how cells function. They send distress signals back to the nucleus that alter gene expression. These messages push cells toward inflammation, stress, and survival pathways that your body normally keeps quiet. Even more concerning, changes in mitochondrial shape — too much splitting (fission) and not enough merging (fusion) — appear in both aging and cancer. These shifts support tumor growth, help cancer cells spread, and make some treatments less effective. Aging mitochondria increase the risk of: brain fogmuscle fatigueslower recoveryheart strainmetabolic slowdowncancer-friendly environments Mitochondria sit at the center of how we age. Why “Mitochondrial Booster” Supplements...

    13 min
  8. Urolithin A - Mitochondrial Miracle in the Petri Dish

    13/11/2025

    Urolithin A - Mitochondrial Miracle in the Petri Dish

    Urolithin A: What It Is, How It Works, and Why Your Gut Decides EverythingBy Dr. Terry Simpson Most people hear the name Urolithin A and think it belongs in a commercial about prostate health. It sounds like something a man named “Gary, 62,” would talk about while fishing. But Urolithin A has nothing to do with plumbing. Instead, it sits at the center of a new wave of longevity science focused on how our cells clean up old, broken parts. As we age, our mitochondria—the tiny power centers inside our cells—start to slow down. They build up damage and stop working well. Eventually, this pile-up makes us lose strength and energy. That’s where Urolithin A comes in. It helps switch back on a process called mitophagy, which is basically the cell’s recycling program for old mitochondria. Where Urolithin A Really Comes FromYou cannot eat Urolithin A directly. Instead, your body makes it when your gut bacteria break down special plant compounds called ellagitannins. These are found in foods like: pomegranateswalnutsberriesgreen tea (yes, really) Green tea is usually known for its catechins, but it also contains ellagitannins like strictinin. After you drink it, your gut bacteria break these tannins apart and create ellagic acid, which can later turn into Urolithin A. However, this only works if you have the right microbes. And here’s the surprising part: Most people do not. Studies show that only 12% to 40% of adults naturally produce Urolithin A from food. Everyone else makes little to none because their gut bacteria simply aren’t built for the job. How Your Gut Decides EverythingYour microbiome—the community of bacteria living in your digestive system—decides whether you make Urolithin A or not. People who produce Urolithin A usually have: more diverse gut microbesspecial bacteria like Enterocloster and Gordonibacterthe right genes inside those microbes to do the chemical conversion People who don’t produce it (called “metabotype zero”) lack those bacteria or the gene pathways needed. Eating more pomegranates or drinking more green tea does not fix this. No diet, including keto or Mediterranean, has been shown to turn a non-producer into a producer. This is why two people can eat the same food, and only one makes Urolithin A. What Urolithin A Does in HumansIn older adults, researchers have tested Urolithin A supplements for up to 4 months. These studies show several encouraging results: muscle endurance improvesinflammation markers decreasemitochondrial health markers look better Even so, there are limits. Trials show no meaningful improvement in: walking distanceATP (cellular energy) productionoverall physical function So the biology looks better, but major clinical outcomes have not changed. What Happens in the Lab (But Not Yet in Humans)Scientists also study Urolithin A in senescent cells—cells that have stopped dividing but still cause inflammation. In the lab, Urolithin A can: reduce senescence markerscalm inflammatory signalsrestore mitophagyimprove oxidative stresseven strengthen circadian rhythms inside aging cells All of this sounds exciting. However, these findings are from cell culture, not humans. They give us clues, not guarantees. Food vs SupplementsYou

    11 min
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Fork U(niversity) Not everything you put in your mouth is good for you. There’s a lot of medical information thrown around out there. How are you to know what information you can trust, and what’s just plain old quackery? You can’t rely on your own “google fu”. You can’t count on quality medical advice from Facebook. You need a doctor in your corner. On each episode of Your Doctor’s Orders, Dr. Terry Simpson will cut through the clutter and noise that always seems to follow the latest medical news. He has the unique perspective of a surgeon who has spent years doing molecular virology research and as a skeptic with academic credentials. He’ll help you develop the critical thinking skills so you can recognize evidence-based medicine, busting myths along the way. The most common medical myths are often disguised as seemingly harmless “food as medicine”. By offering their own brand of medicine via foods, These hucksters are trying to practice medicine without a license. And though they’ll claim “nutrition is not taught in medical schools”, it turns out that’s a myth too. In fact, there’s an entire medical subspecialty called Culinary Medicine, and Dr. Simpson is certified as a Culinary Medicine Specialist. Where today's nutritional advice is the realm of hucksters, Dr. Simpson is taking it back to the realm of science.

Information

  • Creator
    Terry Simpson
  • Years Active
    2018 - 2026
  • Episodes
    112
  • Rating
    Explicit
  • Copyright
    © Copyright 2026 Terry Simpson
  • Show Website
    Fork U with Dr. Terry Simpson

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