The Energy Code

Dr. Mike Belkowski

The Energy Code is your blueprint for unlocking limitless vitality at the cellular level. Hosted by Dr. Mike Belkowski, this podcast dives deep into the science of your mitochondria—the true engines of health and energy. From light, water, and magnetism to groundbreaking molecules and lifestyle upgrades, each episode decodes the most effective strategies to strengthen your “Mitochondrial Matrix.” If you’re seeking cutting-edge science, practical tools, and proven methods to optimize your body and mind, you’ve just cracked the code. Check out these sources: www.biolight.shop – Instagram @biolight.shop – YouTube BioLight

  1. 16 HR AGO

    Decoding Subtle Energy & BioGeometry w/ Nico Martens

    Most people think of health as a game of chemistry and calories. But there is a hidden layer to reality that governs how your biology functions before a single molecule ever moves: subtle energy.   In this episode, Nico Martens joins Dr. Mike to pull back the curtain on BioGeometry and vibrational physics. We explore how "The Card", which is a simple-looking tool engraved with sacred geometric symbols, can objectively alter the taste of wine and the energetic quality of a room by tapping into the "unseen" world. Nico explains why our modern environments are "energetically toxic" and how we can use the principles of BioGeometry to harmonize our homes, mitigate EMF stress, and support mitochondrial vitality at a quantum level.   Then comes the shift. We move from theory to application, discussing how these subtle forces interact with our physical structure. From clearing the "energetic memory" of hotel rooms to using geometric shapes to balance the body's internal systems, this episode provides a blueprint for tapping into the next evolution of human health.   Key Quotes From Nico Martens: “Subtle energy are really all these energies that most of us cannot perceive with our regular senses, but that actually run this entire reality.”   “If you go down to the base level and you break it all the way down, there is no physical matter. There's only vibration.”   “Your environment is either feeding you or it's depleting you.”   “BioGeometry is the language of nature’s design system.”   “The answers to all the challenges we face in the physical world... lay in the unseen.”   Key Points: The Reality of Subtle Energy: Everything in our physical world is built upon a foundation of energy, vibration, and frequency; if you break matter down to its base level, "physical" matter disappears into vibration.   BioGeometry Explained: A science that uses the energy of shapes to balance biological energy systems; it is effectively "tuning" the environment to support life.   The "Wine Test": How specific geometric symbols on a card can change the molecular structure and taste of liquids by harmonizing the subtle energy field.   EMF & Modern Toxicity: Why our current technology creates "discordant" frequencies that stress our mitochondria and how geometry can "harmonize" these signals.   The Power of "The Card": A tool used by practitioners to clear hotel rooms of "energetic debris" and improve the quality of food and water instantly.   Biological Balancing: Using specific shapes to center the body’s energy, helping to mitigate the "cellular noise" of the modern world.   Episode Timeline: 0:00–4:30 — The Wine Story: How Nico used "The Card" to change Dr. Mike’s wine; intro to Nico’s background. 4:31–15:40 — Nico’s Journey: From the physical world to the quantum; meeting in a creek in Tennessee. 16:40–21:15 — Defining Subtle Energy: Beyond the five senses; why Tesla was right about vibration. 21:30–35:20 — What is BioGeometry? The science of shape, the "BG3" centering quality, and nature’s design language. 35:35–45:10 — Environmental Stress: How EMFs and "dirty" energy impact mitochondrial health and the "Mitochondrial Matrix." 45:25–55:00 — Practical BioGeometry: Harmonizing your home and why your environment is either feeding or depleting you. 55:10–01:10:30 — "The Card" Deep Dive: How it works, what the symbols mean, and how to use it in daily life. 01:10:45–01:30:33 — The Unseen World: Why the next evolution of humanity lies in tapping into these subtle dimensions; closing thoughts. Where to learn more from & about Nico Martens: IG: @nicomartens76 Nico Martens Coaching The Wellness Enterprise - Dr. Mike's #1 recommendations:   Deuterium depleted water: Litewater (code: DRMIKE) EMF-mitigating products: Somavedic (code: BIOLIGHT) Blue light blocking glasses: Ra Optics (code: BIOLIGHT) Grounding products: Earthing.com   -   Stay up-to-date on social media: Dr. Mike Belkowski: Instagram LinkedIn   BioLight: Website Instagram YouTube Facebook

    1h 31m
  2. 1 DAY AGO

    A Blue Dye vs. Sudden Blindness: How Methylene Blue Saved Vision After Optic Nerve Trauma

    Most people think head trauma equals concussion. But there’s a silent thief that can ride along with blunt-force injury and steal something just as life-altering: vision.   In this Deep Dive, we break down traumatic optic neuropathy (TON): how shockwaves and shearing forces injure the optic nerve at a bony choke point (the optic canal), then trigger swelling, compartment-syndrome-like pressure, ischemia, and a vicious “cellular riot” that kills neighboring neurons in the days that follow. The brutal reality: traditional options like steroids, surgical decompression, or “observation” often lack strong evidence for reliably saving sight.   Then comes the twist. We review a 2025 peer-reviewed rat study (21-day follow-up) testing methylene blue (MB)immediately after optic nerve crush. MB acts like an electron “bypass road” in the mitochondrial chain, helping keep ATP online when parts of the system are damaged, while also inhibiting the trauma-driven iNOS nitric-oxide flood that creates destructive peroxynitrite. Functionally, the study shows striking preservation of retinal signaling and retinal ganglion cell function, suggesting that immediate metabolic support may blunt secondary degeneration during the “golden window” after injury. - Article Discussed in Episode: Neuroprotective Effect of Methylene Blue in a Rat Model of Traumatic Optic Neuropathy - Key Quotes From Dr. Mike: “There’s a silent thief that often rides shotgun with these head injuries… your vision.” “The optic nerve… is basically the data cable connecting the camera of your eye to the hard drive of your brain.” “Methylene blue… is for all intents and purposes, artificial respiration for the cell’s engine.” “Methylene blue… can act like a temporary bypass road for the bucket brigade.” “You support the energy, you save the structure, you save the function.” - Key points TON can steal vision after blunt trauma even without a direct eye puncture; the optic nerve is a vulnerable “data cable” through a tight bony tunnel (optic canal). The most devastating loss often comes from secondary degeneration: swelling inside the canal raises pressure, chokes blood flow (ischemia), and triggers a cascading “cellular riot.” Standard care is frustratingly limited: steroids, decompression surgery, or observation are described as controversial with limited hard evidence of superiority. There’s a “golden window” (roughly 14–30 days) where tissue is struggling but not fully dead—yet medicine lacks reliable tools to stop the cascade. Methylene blue is not a random supplement: it’s on the World Health Organization list and has long clinical use (e.g., methemoglobinemia). Mechanism #1: MB acts as an electron cycler, bypassing damaged complexes to keep electron flow and ATP production going during ischemic stress. Mechanism #2: MB inhibits iNOS, helping shut down the runaway nitric-oxide surge that forms peroxynitrite, a highly destructive oxidant. In the rat optic nerve crush model, MB given immediately (and repeated doses over 24 hours) produced major functional preservation on ERG measures (especially inner-retina processing signals) and strong evidence of retinal ganglion cell signal preservation, aligning with better structural survival at 21 days. - Episode timeline  1:38–3:38 — TON explained: vision loss after trauma + why medicine feels helpless; introduce MB as the twist 4:01–5:57 — The study: 2025 rat model, what they tested and why MB fits the Energy Code framework 6:11–10:52 — Mechanism and injury cascade: optic canal choke point → swelling → ischemia → secondary degeneration + “cellular riot” 11:07–14:45 — Incidence, current options (steroids/surgery/observation), and the golden window 15:15–20:55 — Why MB: WHO essential medicine, mitochondrial bypass + iNOS/NO “poison valve” control 21:34–24:49 — Methods: optic nerve crush model, groups, dosing (2 mg/kg), timing (0–24h dosing), 21-day follow-up 25:02–31:35 — Results: ERG recovery (B-wave, OPs) + PHNR “complete preservation” of RGC function 32:04–35:17 — Stress testing (pattern ERG) + histology cell counts confirm structure matches function 35:19–38:48 — Human relevance + practicality: animal-to-human caution, but shared mechanisms; preparedness framing + close/CTA - Dr. Mike's #1 recommendations:   Deuterium depleted water: Litewater (code: DRMIKE) EMF-mitigating products: Somavedic (code: BIOLIGHT) Blue light blocking glasses: Ra Optics (code: BIOLIGHT) Grounding products: Earthing.com   -   Stay up-to-date on social media: Dr. Mike Belkowski: Instagram LinkedIn   BioLight: Website Instagram YouTube Facebook

    39 min
  3. 2 DAYS AGO

    Cancer’s Mitochondria Hack: The ‘Second Genome’ and the Epigenetic Software Update That Makes Tumors Adapt

    We all know the common saying: “the mitochondria is the powerhouse of the cell.” But this Deep Dive flips that idea on its head. Instead of a simple battery, mitochondria behave like a second genetic system with its own DNA and its own “software layer” of control.   Using a brand-new January 2026 review on mitochondrial epigenetic mechanisms in cancer by authors from University of Pisa, we explore how tumors hack mitochondrial methylation, DNA packaging, and non-coding RNAsto either floor the gas (energy production for rapid growth) or slam the brakes (metabolic dormancy for survival and metastasis). Then it gets even stranger: mitochondria can send RNA and metabolites that influence the nucleus, while the nucleus sends enzymes and RNAs back into mitochondria—creating a two-way power struggle cancer exploits.   The big takeaway: cancer isn’t only a “mutation problem.” It’s also a reprogramming problem, which opens new doors for diagnostics and therapies designed to target the mitochondrial “operating system” directly. - Article Discussed in Episode: Mitochondrial epigenetic mechanisms in cancer: an updated overview - Key Quotes From Dr. Mike: “What if the powerhouse isn’t just a battery… it’s actually more like an alien spacecraft docked inside us, running its own separate operating system.” “Think of the DNA sequence as your computer hardware. Epigenetics is the software.” “Cancer is when that symbiosis turns into a power struggle.” “Cancer has figured out how to hack it." “Maybe, just maybe, the key to curing cancer isn’t just poisoning the cell, it’s about restoring that ancient peace treaty.” - Key points The “powerhouse” metaphor is incomplete: mitochondria act like a semi-independent system with a second genome and complex regulation. Mitochondrial DNA is small but vital (circular, bacterial-like), supporting the idea of an endosymbiotic origin. The review focuses on epigenetics: not changing DNA letters, but changing how genes are read via methylation “switches.” A long-running debate is framed as resolved: mitochondrial DNA can be methylated by enzymes that enter mitochondria, allowing gene silencing similar to the nucleus. Mitochondria also regulate access to their DNA through packaging proteins (a “tape/dimmer switch” controlling expression and energy output). Gas pedal: hypomethylation in key control regions (like the D-loop) to ramp up output for growth. Brake: hypermethylation to suppress replication and shift toward dormancy during hostile transitions (like metastasis). Cancer uses two strategies depending on context: Non-coding RNAs become “regulatory managers”: sense/antisense balance can be disrupted so tumors lose “stop signals,” and restoring the “good twin” can trigger selective tumor cell death in models. The future direction is precision oncology: using stable mitochondrial methylation/RNA signatures for screening (blood/urine signals) and designing therapies that specifically target mitochondrial epigenetic machinery. - Episode timeline  0:19 — Intro sting + the “powerhouse of the cell” meme setup 0:55 — Reframe: mitochondria as an “operating system” that cancer can hack 1:35 — The January 2026 review + mission: understand “mitoepigenetics” 2:13 — The “second genome”: mtDNA basics + endosymbiotic origin 3:26 — Epigenetics explained: software vs hardware; methylation as gene switches 4:40 — Debate resolved: mtDNA methylation exists; enzymes can tag/silence mtDNA 5:02 — mtDNA packaging (TFAM “tape”) + the mitochondrial “dimmer switch” idea 5:57 — Cancer’s two modes: gas vs brake strategies 6:14 — Gas pedal example: D-loop hypomethylation → increased output for growth 7:23 — Brake example: hypermethylation → reduced mitochondria + metabolic dormancy (metastasis survival) 8:40 — Drug resistance angle: methylation changes that help cells evade death triggers 9:41 — Non-coding RNAs: sense vs antisense “RNA twins” and the loss of brakes 11:26 — Viral hacking example: HPV-style mitochondrial reprogramming framing 12:30 — Therapeutic concept: reintroducing the “good twin” → selective apoptosis in models 13:25 — Circular RNAs and micro-RNAs: stable signals; cancer-type-specific roles 16:25 — Mitonuclear crosstalk: two-way signaling; mitochondria can influence nuclear epigenetics 18:55 — What this enables: diagnostics (blood/urine), mito-targeted therapies, gene-editing concepts 20:33 — Big metaphor: restoring the “peace treaty” (symbiosis) vs hacking - Dr. Mike's #1 recommendations:   Deuterium depleted water: Litewater (code: DRMIKE) EMF-mitigating products: Somavedic (code: BIOLIGHT) Blue light blocking glasses: Ra Optics (code: BIOLIGHT) Grounding products: Earthing.com   -   Stay up-to-date on social media: Dr. Mike Belkowski: Instagram LinkedIn   BioLight: Website Instagram YouTube Facebook

    23 min
  4. 3 DAYS AGO

    The Taurine ‘Master Switch’: How a Common Mold Toxin Wrecks the Gut (and How Cells Rebuild from the Bottom Up)

    In this The Energy Code Deep Dive, we start with a topic that sounds like a prank: piglets + taurine. But it turns into a surprisingly universal lesson about how gut damage happens and how recovery actually works.   We walk through research using piglets as a model for intestinal injury caused by DON (deoxynivalenol), a Fusarium mold toxin commonly found in grain contamination. DON doesn’t just irritate the gut. It collapses the mucus barrier(goblet cells and MUC2), breaks tight junction “zippers” (ZO2, occludin, claudin), and triggers apoptosis (BAX up, caspase-3 up, BCL-2 down). Underneath it all is the real root: mitochondrial failure—swollen, damaged cristae, and ATP levels dropping.   Then comes the twist: taurine doesn’t “patch the wall.” It restores the power, revives mitochondrial function (and antioxidant defenses like SOD2), and appears to reactivate the PGC-1 → NRF1/NRF2 axis—the factory-manager-and-foremen system that turns the repair program back on. The big takeaway: many “structural” problems may secretly be energy problems. - Article Discussed in Episode: Taurine ameliorates deoxynivalenol-induced intestinal injury in piglets: Restoration of mitochondrial function linked to the PGC1α-NRF1/2 axis - Key Quotes From Dr. Mike: “We’re trying to fix the cracks in the wall, but maybe we should be checking the fuse box in the basement.” “If mitochondria are like little power plants, mitophagy is like the maintenance crew that removes broken generators before they start smoking up the whole city.” “In the DON toxin group… the mitochondria aren’t just stressed. They’re swollen… the cristae are broken.” “Taurine didn’t fix the machines itself. It walked into the office and woke up the manager.” “Taurine just flipped the breaker back on.” - Key points Piglets are the model; the biology is the message—the episode uses the gut as a window into a universal cellular fight. DON (a Fusarium mycotoxin) acts like a gut saboteur: it doesn’t just inflame tissue, it disrupts the cell’s energy system. The first visible hit is the mucus barrier: goblet cells drop, MUC2 production falls, and the “moat around the castle” drains. The next hit is the tight junction barrier: ZO2/occludin/claudin markers fall—your intestinal “zipper” loses teeth, turning into a literal leak. Cells enter a panic state and trigger apoptosis: BAX/caspase-3 rise, BCL-2 drops—a death order goes out. The root cause is mitochondrial dysfunction: swollen mitochondria, damaged cristae, ATP depletion—the factory loses power. Taurine reverses the cascade: preserves goblet cells, restores tight junctions, reduces apoptosis signals, and improves mitochondrial function + antioxidant support (SOD2). Mechanistic “why”: taurine appears to reactivate the PGC-1 / NRF1 / NRF2 repair axis, essentially waking up the mitochondrial rebuild program—energy first, structure second. - Episode timeline  00:00–01:20 — Cold open: piglets + taurine + why this isn’t an energy drink episode 01:20–03:00 — The villain: DON mycotoxin (Fusarium on grains) + why the model matters 03:00–05:00 — Barrier layer #1: goblet cells + MUC2 mucus “moat” collapses; taurine restores it 05:00–06:40 — Barrier layer #2: tight junction “zipper” (ZO2/occludin/claudin) breaks; taurine reseals it 06:40–08:40 — Cellular crime scene: apoptosis lights up (TUNEL), BAX/caspase-3 up, BCL-2 down; taurine flips the death balance 08:40–10:50 — The engine room: mitochondria swell, cristae break, ATP drops; taurine restores structure + ATP + SOD2 10:50–13:10 — The master switch: PGC-1 → NRF1/NRF2 axis as manager/foremen/blueprints; DON silences it, taurine reactivates it 13:10–15:00 — Big takeaway: many conditions may be “energy problems” first; closing reflections + sign-off - Dr. Mike's #1 recommendations:   Deuterium depleted water: Litewater (code: DRMIKE) EMF-mitigating products: Somavedic (code: BIOLIGHT) Blue light blocking glasses: Ra Optics (code: BIOLIGHT) Grounding products: Earthing.com   -   Stay up-to-date on social media: Dr. Mike Belkowski: Instagram LinkedIn   BioLight: Website Instagram YouTube Facebook

    17 min
  5. 4 DAYS AGO

    Methylene Blue + Light vs. Superbugs: A Surgical-Style Kill Switch for Antibiotic-Resistant Infections?

    In this Energy Code Deep Dive, we review a real-world, high-stakes study that sounds like sci-fi: using methylene blue + a specific wavelength of light to kill antibiotic-resistant bacteria. The clinical backdrop is pediatric perforated appendicitis, where bacteria can leak into the abdomen and lead to serious infections, long hospital stays, and heavy IV antibiotic use.   The paper tests photodynamic therapy (PDT): add a light-sensitive dye (methylene blue), shine 665nm light, and generate reactive oxygen species that inflict broad oxidative damage on microbes—often regardless of classic antibiotic resistance mechanisms. The results are striking for several major pathogens, with huge log reductions for E. coli and Streptococcus anginosus group, and more variable results for Pseudomonas aeruginosa.   We also keep it grounded: it’s in vitro, not yet a clinical protocol, and it didn’t test everything you’d want (biofilms, polymicrobial mixtures, anaerobes). But as a proof-of-concept, it’s a strong argument that light can be a precise medical tool—when parameters are engineered, not guessed. - Article Discussed in Episode: Photodynamic therapy with methylene blue effectively kills antibiotic resistant bacteria from pediatric patients with perforated appendicitis - Key Quotes From Dr. Mike: “Photodynamic therapy doesn’t rely on the same mechanisms as antibiotics, so it can work even when bacteria are resistant.” “You take a dye that’s light sensitive… add it to the bacteria, then you shine a specific wavelength of light.” “Methylene blue is the match, the light is the strike.” “A 6-log reduction is a millionfold reduction… basically a wipeout in this kind of lab setup.” “The key takeaway is resistance didn’t protect bacteria from this approach in most cases.” - Key points The study targets a big clinical problem: perforated appendicitis in kids → intra-abdominal infection risk, long hospitalization, heavy antibiotic exposure. The intervention is photodynamic therapy (PDT): methylene blue + 665nm light → reactive oxygen species that damage bacteria. PDT doesn’t rely on standard antibiotic mechanisms, so it can work even when bacteria are antibiotic-resistant. Methods: bacteria isolated from peritoneal fluid samples (30 patients) and tested under 4 conditions: control, dye-only, light-only, dye+light. Parameters matter: 665nm laser via fiber optic; low fluence rate (4 mW/cm²), total fluence 7.2 J/cm², 30 min; methylene blue 300 mcg/mL. Most prevalent organisms included E. coli, Strep anginosus group, Bacteroides fragilis, Pseudomonas aeruginosa; polymicrobial infections were common. Results: ~5.86–5.91 log₁₀ reductions for E. coli and Strep anginosus group (massive kill); Pseudomonas showed smaller, variable reductions (~2.23 log₁₀). Limitations: in vitro, planktonic monocultures (not biofilms/mixed communities), anaerobes not tested in PDT setup, and parameter optimization still needed—yet the proof-of-concept is very promising for a localized surgical adjunct. - Episode timeline  0:19 — Intro sting + welcome back 0:32 — Topic hook: methylene blue + light vs antibiotic-resistant bacteria 1:06 — Clinical context: pediatric perforated appendicitis → infections, IV antibiotics, long stays 1:49 — PDT explained simply (dye + light → ROS; works beyond antibiotic resistance) 2:39 — Study design: 30 patient samples; isolate bacteria; 4 test conditions 3:13 — Technical parameters: 665nm laser/fiber optics; dosing details; why specs matter 3:50 — What bacteria were found; polymicrobial reality + resistance common 4:36 — Results: major kills for E. coli & Strep; Pseudomonas more variable 5:13 — Quick “log reduction” translation for normal humans 6:16 — Resistant vs susceptible strains: resistance didn’t protect (most cases) 6:54 — Big vision: localized surgical adjunct via laparoscopic illumination 7:24 — Limitations: in vitro, monocultures, no biofilms/mixed species, anaerobes not tested, optimization needed 8:19 — BioLight tie-in (light as precise tool; don’t wing it; specs matter) 9:29 — One-sentence takeaway - Dr. Mike's #1 recommendations:   Deuterium depleted water: Litewater (code: DRMIKE) EMF-mitigating products: Somavedic (code: BIOLIGHT) Blue light blocking glasses: Ra Optics (code: BIOLIGHT) Grounding products: Earthing.com   -   Stay up-to-date on social media: Dr. Mike Belkowski: Instagram LinkedIn   BioLight: Website Instagram YouTube Facebook

    11 min
  6. 5 DAYS AGO

    Urolithin A: The Human Data on ‘Anti-Aging’ (Mitophagy, Inflammation, Muscle — What Actually Changes)

    In this Energy Code Deep Dive, Dr. Mike Belkowski and moderator Don Bailey break down a 2024 systematic review, “Targeting Aging With Urolithin A in Humans," that focuses on human supplementation studies, not “eat pomegranate and hope.”    You’ll learn what Urolithin A is (and why your gut bacteria can make results wildly inconsistent), why it’s tied to “geroprotection,” and what the clinical evidence actually supports so far: dose-dependent anti-inflammatory signals, changes in mitochondrial/autophagy gene markers, and some improvements in strength/endurance — with a reality check on what didn’t move (ATP max, broad physical function, microbiome composition, body comp, cardiovascular markers in short windows).   Bottom line: promising, practical, but still early. - Article Discussed in Episode: Targeting aging with urolithin A in humans: A systematic review - Key Quotes From Dr. Mike & Don: “It’s like giving two people the same coffee beans, but one of them doesn’t own a coffee grinder!” "It may be improving the ‘quality control and efficiency settings’ more than raw peak horsepower.“ “So it’s like tuning the car so it runs smoother; not necessarily making the top speed higher.” "It’s not a ‘lose 20 pounds and become a triathlete’ pill.” - Key points Urolithin A is a gut-derived metabolite from ellagic acid foods (pomegranate, walnuts, berries), but many people don’t convert well. So food intake ≠ reliable levels. Supplementation “skips the gut lottery” and produces higher, more consistent plasma levels than food sources. The systematic review included 5 human studies / 250 healthy participants with 10–1000 mg/day for 28 days to 4 months. Biggest consistent theme: dose-dependent anti-inflammatory effects (some markers improve more at 1000 mg/day over 4 months). Mitochondria story is nuanced: it may improve gene expression signatures related to mitochondrial activity, autophagy, and fatty-acid oxidation—more “quality control” than peak power. What it didn’t reliably do: increase maximal ATP production, consistently boost biogenesis/dynamics markers, change gut microbiota composition, or meaningfully affect body metrics/cardiovascular outcomes in short trials. Muscle outcomes: some gains in specific strength/endurance measures (e.g., torque metrics; certain fatigue tests), but not universal (e.g., handgrip and broad function didn’t consistently improve). Safety in these studies looked clean (no serious adverse events attributed), but the overall conclusion remains: promising—but the human aging evidence is still young and needs longer/larger trials. - Episode timeline  0:27 — Welcome + urolithin A is trending + episode topic 0:45 — Don’s moderator frame: “does it work / worth it?” 1:04 — Paper ID (2024 systematic review; human supplementation) 1:25 — What urolithin A is + “gut lottery” 2:02 — Why supplement (higher/consistent levels vs food) 2:24 — Why it matters: mitophagy / healthier aging angle 3:05 — What’s included (5 studies, 250 people; 10–1000 mg; 28d–4mo) 3:34 — What improved (inflammation signals, gene markers, some strength/endurance) 4:06 — What didn’t (max ATP, microbiome, broad health/body metrics) 7:02 — Dosing/PK basics (peak ~6h; half-life ~17–22h; plateau ~7d) 7:40 — Safety summary 8:06 — Practical take + “promising but early” conclusion - Dr. Mike's #1 recommendations:   Deuterium depleted water: Litewater (code: DRMIKE) EMF-mitigating products: Somavedic (code: BIOLIGHT) Blue light blocking glasses: Ra Optics (code: BIOLIGHT) Grounding products: Earthing.com   -   Stay up-to-date on social media: Dr. Mike Belkowski: Instagram LinkedIn   BioLight: Website Instagram YouTube Facebook

    11 min
  7. 6 DAYS AGO

    Chemobrain Breakthrough? Near-infrared Light Pilot Study Shows Striking Cognitive Gains

    Welcome to the first Energy Code Deep Dive—daily research reviews translated into real life. Dr. Mike Belkowski and co-host Don Bailey break down a brand-new (Jan 3, 2026) pilot study on transcranial photobiomodulation (tPBM) for chemobrain (cancer-related cognitive impairment).   We define what chemobrain actually feels like, why there aren’t many proven treatments, and why researchers are exploring 810nm brain-directed light + an intranasal component to support mitochondrial energy (cytochrome c oxidase/ATP), inflammation balance, blood flow, and repair signaling.   Then we walk through the real-world clinical cohort (31 women), the protocol (weekly sessions, ~20 minutes, 10+ sessions), and the eye-opening outcomes: 29/31 improved, average cognitive scores rose dramatically, and a meaningful percentage normalized. We also keep it honest—small sample, retrospective design, no control group—so you know what’s promising now and what still needs randomized trials. - Article Discussed in Episode: Transcranial photobiomodulation for the treatment of chemobrain: new perspectives from a pilot study - Key Quotes From Dr. Mike: “It’s like your brain’s running 30 browser tabs and somebody started a video call in the background.” “Think of it like giving your brain cells a more efficient ‘charge cycle,’ not by caffeine, but by improving cellular energy production.” “This is why device specs aren’t nerd trivia. They’re the difference between a protocol and a placebo.” “This pilot study suggests that transcranial photobiomodulation may meaningfully improve chemobrain symptoms… but we still need larger controlled trials.” - Key points Chemobrain is real: attention, processing speed, verbal fluency, executive function — often lingering for years and impacting daily life. The study reviewed a Jan 3, 2026 pilot exploring tPBM as a potential supportive treatment when proven options are limited. Mechanism focus: light targets mitochondrial function (cytochrome c oxidase → ATP), with downstream effects on inflammation, blood flow, and repair signaling. Cohort: 31 women, average age ~52, post-chemo cognitive impairment; cognition tracked via FACT-Cog. Protocol: 810nm transcranial + intranasal, ~20 min/session, weekly, 10+ sessions; some also used whole-body PBM. Why 810nm: penetration matters; modeling suggests near-optimal depth to reach cortical targets; intranasal may help access harder-to-reach regions. Results were striking: average score improved from ~63 to ~101; 29/31 improved; ~29% normalized into typical range. Limitations & takeaway: retrospective + no control group (can’t rule out time/placebo), but the effect size supports moving toward larger randomized trials and reinforces that parameters/device specs matter. - Episode timeline  00:00 – Welcome to the first Energy Code Deep Dive + Don’s role as the “question-asker” 01:30 – What is chemobrain (symptoms + what it feels like day-to-day) 04:00 – Why treatment options are limited (the “brutal part”) 05:30 – What transcranial photobiomodulation is (plain-English translation) 07:30 – Biology: cytochrome c oxidase, ATP, inflammation, blood flow, repair signaling 10:00 – Study design + who they studied (31 women, France, post-chemo, FACT-Cog) 12:30 – Real-life impacts (reading, admin work, conversations, driving, fatigue, sleep) 14:30 – Protocol details (810nm, intranasal, weekly 20 min, 10+ sessions, some whole-body) 17:00 – Why 810nm + why intranasal (penetration + access) 19:00 – Results (63 → 101, 29/31 improved, ~29% normalized, QoL changes) 21:30 – Mood/anxiety/depression findings + interpretation 23:00 – Mechanisms: chemo injury pathways vs PBM supportive pathways 25:30 – Limitations (small sample, retrospective, no control, can’t split brain vs whole-body) 27:30 – Safety notes + “what do I do with this?” (talk to clinician; parameters matter) 29:00 – One-sentence takeaway + close (“Protect your energy / mitochondria”) - Dr. Mike's #1 recommendations:   Deuterium depleted water: Litewater (code: DRMIKE) EMF-mitigating products: Somavedic (code: BIOLIGHT) Blue light blocking glasses: Ra Optics (code: BIOLIGHT) Grounding products: Earthing.com   -   Stay up-to-date on social media: Dr. Mike Belkowski: Instagram LinkedIn   BioLight: Website Instagram YouTube Facebook

    11 min
  8. 29 JAN

    The Mitochondria Fix for Alzheimer’s, Parkinson’s & ALS (Therapies Moving Beyond Symptoms)

    Neurodegeneration can feel like an unstoppable train, where treatment is often about managing symptoms instead of fixing what’s breaking upstream. In this episode of The Energy Code, Dr. Mike Belkowski and co-host Don Bailey unpack a 2025 review on mitochondrial dysfunction as a common thread across major neurodegenerative diseases and why this “cellular infrastructure problem” may be the leverage point researchers are finally targeting.    You’ll get clear, memorable analogies for how energy production fails (oxidative phosphorylation), why Complex I dysfunction shows up so often in Parkinson’s, and why ROS isn’t “bad” until it becomes a wildfire.  From there, they walk through mitochondrial quality control (fission, fusion, and mitophagy) plus the underrated issue of mitochondrial transport in long neurons.    Finally, we break down what’s being explored right now: mitochondria-targeted antioxidants, metabolic interventions, photobiomodulation, biogenesis strategies (PGC-1α), and emerging frontiers like mitochondrial transplantation and gene editing—with a grounded take on what’s promising vs. what’s still early. - Key Points Neurodegeneration is increasingly being viewed as an upstream mitochondrial dysfunction problem and not just a symptom-management problem.  The brain is an extreme energy consumer, so neurons are uniquely vulnerable when ATP production drops.  Oxidative phosphorylation failure reduces cellular power and sets the stage for degeneration. In Parkinson’s, Complex I impairment is a recurring theme → less ATP + more oxidative stress. ROS isn’t inherently bad — it’s signaling vs “wildfire” oxidative stress when defenses get overwhelmed.  Mitochondrial “quality control” (fission, fusion, mitophagy) is central; breakdown accelerates damage.  Neurons depend on mitochondrial transport down long axons; transport failure can starve synapses first.  Emerging interventions include mitochondria-targeted antioxidants, biogenesis/repair pathways, and mitochondria-relevant trials (including photobiomodulation) promising, but still evolving. - Key Quotes From Dr. Mike “Fission is like splitting dough into separate pizzas. Too much fission is like cutting everything into tiny crumbs.”  “Too little fission is like refusing to separate the burnt part… it ruins the whole batch.”  “This is the recycling program… Tag it, bag it, take it out.”  “PGC-1 alpha is like the head contractor for building new power plants.”  “If demand doubles, you can’t keep running on the same number of servers… you need more infrastructure.” - Article Referenced in Episode: Mitochondrial-based therapies for neurodegenerative diseases: a review of the current literature - Episode Timeline 00:00 — Cold open + show intro (“The Energy Code” mission / mitochondrial matrix)  00:45 — The “unstoppable train” problem: symptoms vs upstream causes  02:00 — Why mitochondria matter in the brain (energy hog / “city that never sleeps”)  04:00 — Quick definitions: Alzheimer’s vs Parkinson’s vs ALS  05:30 — Oxidative phosphorylation explained (dam/turbine analogy)  08:00 — Parkinson’s spotlight: Complex I disruption + downstream ROS  10:00 — ROS with nuance: signaling vs oxidative stress + antioxidant balance  12:30 — The quality-control trio: fission, fusion, mitophagy (pizza + recycling analogies)  15:30 — Mitochondrial transport in neurons (train tracks: kinesin/dynein)  17:00 — Therapy bucket #1: mitochondria-targeted antioxidants (why targeting matters)  18:30 — “Is it being tested?” clinical trial examples across AD/PD/ALS  20:30 — Photobiomodulation: keep it grounded (flashlight/battery analogy)  22:30 — Biogenesis: PGC-1α + AMPK/SIRT1 + exercise mimetics  25:00 — Frontiers: transplantation, gene therapy/CRISPR, ethics/regulation  28:30 — Five takeaways + closing call-to-action (subscribe/review/share) - Dr. Mike's #1 recommendations:     Deuterium depleted water: Litewater (code: DRMIKE) EMF-mitigating products: Somavedic (code: BIOLIGHT) Blue light blocking glasses: Ra Optics (code: BIOLIGHT) Grounding products: Earthing.com - Stay up-to-date on social media: Dr. Mike Belkowski: Instagram LinkedIn   BioLight: Website Instagram YouTube Facebook

    27 min

About

The Energy Code is your blueprint for unlocking limitless vitality at the cellular level. Hosted by Dr. Mike Belkowski, this podcast dives deep into the science of your mitochondria—the true engines of health and energy. From light, water, and magnetism to groundbreaking molecules and lifestyle upgrades, each episode decodes the most effective strategies to strengthen your “Mitochondrial Matrix.” If you’re seeking cutting-edge science, practical tools, and proven methods to optimize your body and mind, you’ve just cracked the code. Check out these sources: www.biolight.shop – Instagram @biolight.shop – YouTube BioLight

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