Healthy AF | Healthy After 50

Zane Griggs: Health Tips

Healthy AF, the podcast dedicated to helping you live a healthier life after the age of 50, hosted by health and longevity expert Zane Griggs. This show is your guide to achieving optimal health, both physically and mentally. Whether you're in your 20s and want to plan for a healthy future or in your 50s and seeking to catch up, this show could help you improve your nutrition, fitness, and overall well-being. Each week, we'll dive into the latest research and expert advice on topics such as exercise, stress management, and more. We'll discuss practical strategies for maintaining a healthy lifestyle, including how to balance your diet and stay active as you age. Remember, it's never too late to prioritize your health and become Healthy AF! (Hit play to learn more)

  1. Jun 4

    51. The Seed Oil Studies Are Measuring the Wrong Thing

    A new seed oil study may look reassuring at first glance — but what did it actually measure? In this episode, Zane Griggs breaks down a 2025 review of clinical studies on seed oils and explains why short-term improvements in blood markers do not necessarily tell us what is happening inside the mitochondria. You'll learn why the real concern is not only what shows up in a blood draw, but what happens when polyunsaturated fats become incorporated into mitochondrial membranes over time. Zane walks through: what the seed oil review actually found why flaxseed and sesame oil are not the same as soybean or corn oil the difference between blood markers and mitochondrial damage cardiolipin, ATP production, and oxidative stress why 4-HNE and other aldehydes matter how cooking oils change when heated why stored linoleic acid can remain in the body for years why a short-term study may miss a long-term mitochondrial problem how soybean oil consumption has changed over the last century why stable fats may matter more than most people realize Studies: Laurindo et al. Frontiers in Nutrition. 2025;12:1502815 Hulbert AJ et al. Physiological Reviews. 87:1175–1213, 2007 Seebacher F, Brand MD, Else PL, Guderley H, Hulbert AJ, Moyes CD. Physiological and Biochemical Zoology. 83(5):721–732, 2010 UC Riverside / Journal of Lipid Research, November 2025 SNI Global soybean oil consumption data The point is not to ignore studies that show short-term improvements. The point is to understand their limitations and ask whether they are measuring the outcome that actually matters. Download Zane's Free Fit Over 40 Plan: https://free40plan.com   00:00 The seed oil study everyone is going to cite 01:00 What the 2025 review actually measured 02:00 Why flaxseed and sesame oil are different 03:30 The short-term benefits are real — but limited 04:00 The problem with relying on blood markers 05:00 What happens inside the mitochondria 06:00 Cardiolipin and ATP production explained 07:00 The damage the studies did not measure 08:00 Why higher antioxidant activity can be misleading 09:00 The researchers admitted a major limitation 10:00 They did not verify the oil composition 11:00 Why processing, heat, and storage matter 12:00 Fast-food fryer oil is not the same intervention 13:00 How stored linoleic acid remains in the body 15:00 Why the control groups were not truly low-PUFA 16:00 The washout problem no short-term study solves 17:00 Comparing two versions of excess exposure 18:00 How much soybean oil Americans consume 19:00 Physiological need vs modern consumption 21:00 Why the baseline recommendation may already be too high 22:00 The UC Riverside soybean oil research 23:00 Oxylipins, liver health, and mitochondrial function 25:00 Why standard blood tests may miss the damage 26:00 The peroxidation index explained 27:00 Omega-3 vs omega-6 vs monounsaturated fats 28:00 Why more fish oil may not solve the problem 29:00 Saturated fat and mitochondrial membrane stability 31:00 What membrane fluidity actually requires 32:00 Why longer-lived species matter 33:00 What the pro-seed-oil literature can and cannot prove 35:00 Short-term blood improvements vs long-term membrane loading 36:00 What fats Zane recommends using instead 38:00 Why ingredient labels matter 39:00 Final takeaway: read the studies carefully

    41 min
  2. Jun 2

    50. Your Body Is Stuck Burning Fat — And It's Making You Sick (Part 2)

    What happens when your body gets stuck burning fat and can't switch back to glucose? In Part 2, Zane breaks down metabolic inflexibility and explains why being locked into fat-dominant metabolism is not the same as being metabolically healthy. You'll learn how this pattern affects the muscle, heart, and liver — and how elevated fatty acids can impair glucose oxidation, disrupt insulin signaling, and contribute to insulin resistance, fatty liver disease, and heart failure. Start with Part 1 first for the ATP and glucose-efficiency breakdown. 01:00 The metabolic signature of insulin resistance 03:30 Why your body should switch between fat and glucose 06:00 The 3 tissues that reveal metabolic inflexibility 08:30 Why Zane changed his mind about low carb 13:30 The real definition of metabolic inflexibility 15:00 Why elevated blood sugar can happen on keto or carnivore 20:00 Heart failure and metabolic disease 22:00 Why failing hearts become more dependent on fat 25:00 Why glucose is not the problem 29:00 The liver and fatty liver disease 33:00 Fatty liver is a metabolic inflexibility problem 34:00 Why the liver starts producing more glucose 40:00 How liver disease progresses 41:00 The muscle, heart, and liver connection 46:00 How to restore glucose oxidation 47:00 Why high-fat, low-carb can mimic metabolic disease   Kelley, D.E. (2005) "Skeletal muscle fat oxidation: timing and flexibility are everything" Journal of Clinical Investigation — JCI25758 / PMC1159159 Used for: Definition of metabolic inflexibility; experimental elevation of plasma FFAs recreating T2D metabolic signature in skeletal muscle; blunted insulin-stimulated glucose oxidation; impaired suppression of lipid oxidation Ukropcova, B. et al. (2005) "Dynamic changes in fat oxidation in human primary myocytes mirror metabolic characteristics of the donor" Journal of Clinical Investigation — JCI24332 Used for: Formal definition of metabolic inflexibility — insulin-resistant muscle characterized by lower fasting lipid utilization and failure to switch to carbohydrate oxidation in response to insulin Sun, Q., Lopaschuk, G.D. et al. (2024) "Mitochondrial fatty acid oxidation is the major source of cardiac ATP production in heart failure with preserved ejection fraction" Cardiovascular Research, Volume 120 — PMID 38193548 Used for: Direct radiolabeled substrate measurements in HFpEF; suppressed insulin-stimulated glucose oxidation; increased FAO; decreased PDH phosphorylation confirmed in human heart samples; disrupted glucose/fat ATP balance Sun, Q., Karwi, Q.G., Wong, N., Lopaschuk, G.D. (2024) "Advances in myocardial energy metabolism: metabolic remodelling in heart failure and beyond" Cardiovascular Research — PMC11646102 Used for: Comprehensive synthesis — decreased glucose oxidation as universal defect across HFpEF, HFrEF, and diabetic cardiomyopathy; uncoupling of glycolysis from glucose oxidation; FAO increases in HFpEF and diabetic cardiomyopathies Lopaschuk, G.D., Ussher, J.R., Folmes, C.D.L., Jaswal, J.S., Stanley, W.C. (2010) "Myocardial Fatty Acid Metabolism in Health and Disease" Physiological Reviews — PMC3976623 Used for: Fat oxidation dominance in HF, ischemia, and diabetes; uncoupling of glycolysis from glucose oxidation; therapeutic case for reducing FAO and increasing glucose oxidation (quotes carried over from Video 1 for context) Fillmore, N., Jaswal, J.S., Lopaschuk, G.D. (2011) "Uncoupling of glycolysis from glucose oxidation accompanies the development of heart failure" Journal of Molecular and Cellular Cardiology — ScienceDirect Used for: Pharmacological shifting from fat to glucose oxidation improving ATP efficiency; cardiac ischemia and heart failure therapeutic mechanism (context reference from Video 1) Satapati, S. et al. (2011) "Excessive hepatic mitochondrial TCA cycle and gluconeogenesis in humans with nonalcoholic fatty liver disease" Cell Metabolism — PMC3658280 Used for: 50% higher lipolysis in NAFLD; 30% higher gluconeogenesis; 2-fold increase in TCA cycle flux correlated with intrahepatic triglyceride content; increased FFA delivery and oxidation driving gluconeogenesis and oxidative stress Donnelly, K.L., Smith, C.I., Schwarzenberg, S.J., Jessurun, J., Boldt, M.D., Parks, E.J. (2005) "Sources of fatty acids stored in liver and secreted via lipoproteins in patients with nonalcoholic fatty liver disease" Journal of Clinical Investigation — JCI23621 Used for: Fat source breakdown in NAFLD — ~60% from plasma NEFA/adipose lipolysis, ~26% from de novo lipogenesis, ~15% dietary fat; stable isotope methodology confirming adipose lipolysis as primary driver Kolwicz, S.C. Jr. (2021) "Ketone Body Metabolism in the Ischemic Heart" Frontiers in Cardiovascular Medicine — DOI 10.3389/fcvm.2021.789458 Used for: Supporting context on cardiac metabolic flexibility; KD association with worse ischemic outcomes in some models; ketone-glucose suppression dynamic in the ischemic heart

    59 min
  3. May 29

    50. Glucose Is the Most Efficient Fuel | The ATP & Metabolism Science Explained (Part 1)

    What if the most efficient fuel for the human body isn't fat… but glucose? In this episode, Zane Griggs breaks down the actual biochemistry behind ATP production, mitochondrial efficiency, and why cardiologists actively try to shift failing hearts away from fat oxidation and back toward glucose oxidation. This conversation dives deep into the P/O ratio, oxygen efficiency, insulin resistance, heart failure, ketones, metabolic flexibility, and the misunderstood role of glucose in human metabolism. If you've been told that fat is always the superior fuel source, this episode may completely change how you think about metabolism, performance, and energy production. In this episode: Why glucose produces more ATP per oxygen molecule The truth about fat oxidation vs glucose oxidation Why the heart becomes "stuck" burning fat in heart failure The real meaning of metabolic flexibility Why insulin is not the villain it's been made out to be The difference between glycolysis and glucose oxidation Why ketones are not the "super fuel" many claim How mitochondrial inefficiency impacts metabolic disease The connection between diabetes, heart disease, and fuel selection This is Part 1 of a deeper metabolic series exploring insulin resistance, ATP production, glucose metabolism, and how the body actually creates energy. Feeling "off" despite doing everything right? Download Zane's free Metabolic Stress Marker Guide to learn: ✔ what your labs may actually be telling you ✔ why "normal" doesn't always mean optimal ✔ the hidden hormone, thyroid, and metabolic markers most people overlook 👉 Get the free guide: https://zanegriggs.com/freeguide Ready to learn more? Get Zane's Free Over 40 Performance Plan: https://free40plan.com 00:00 Why fuel efficiency matters 01:00 What is ATP and the P/O ratio? 02:00 Why oxygen efficiency changes everything 03:00 Calories vs mitochondrial efficiency 05:00 The engine analogy explained 06:00 The studies comparing glucose, fat, and ketones 08:00 Why glucose creates more ATP than fat 09:00 What happens in heart failure 11:00 Are ketones really a "super fuel"? 13:00 Ketones and mitochondrial uncoupling 14:00 How cardiologists use glucose therapeutically 16:00 Fat oxidation and heart dysfunction 18:00 Diabetes, heart disease, and blocked glucose oxidation 20:00 Why doctors shift patients toward glucose oxidation 22:00 The metabolic flexibility explanation 24:00 Fat oxidation blocking glucose oxidation 25:00 Why insulin is NOT the enemy 27:00 The real driver of insulin resistance 28:00 Key takeaways on glucose vs fat burning 30:00 Preview of Part 2   Mookerjee et al. (2017) "Quantifying intracellular rates of glycolytic and oxidative ATP production and consumption using extracellular flux measurements" Journal of Biological Chemistry — PMC5409486 Used for: Glucose P/O ratio of 2.79; updated theoretical maximum ATP yields Agilent Technologies / Seahorse Bioscience (white paper) "Quantifying ATP Production Rate Using the Seahorse XF Real-Time ATP Rate Assay" Agilent application note 5991-9303EN Used for: P/O ratio range — palmitate 2.45, glucose 2.79–2.86; average cellular P/O of 2.75 Lopaschuk, Ussher, Folmes, Jaswal, Stanley (2010) "Myocardial Fatty Acid Metabolism in Health and Disease" Physiological Reviews — PMC3976623 Used for: All four extended quotes on fat oxidation dominance in heart failure, ischemia, and diabetes; therapeutic strategy of reducing fat oxidation and increasing glucose oxidation Fillmore, Jaswal, Lopaschuk (2011) "Uncoupling of glycolysis from glucose oxidation accompanies the development of heart failure" ScienceDirect / Journal of Molecular and Cellular Cardiology Used for: Quote on pharmacological shifting from fat to glucose oxidation improving ATP efficiency; cardiac ischemia and heart failure Sun, Lopaschuk et al. (2024) "Mitochondrial fatty acid oxidation is the major source of cardiac ATP production in heart failure with preserved ejection fraction" Cardiovascular Research, Volume 120 — PMID 38193548 Used for: HFpEF data — suppressed insulin-stimulated glucose oxidation, increased FAO, decreased PDH phosphorylation in mouse and human samples Kolwicz (2021) "Ketone Body Metabolism in the Ischemic Heart" Frontiers in Cardiovascular Medicine — DOI 10.3389/fcvm.2021.789458 Used for: Ketone P/O ratio ~2.50 vs glucose ~2.58 vs fat ~2.33; ischemic heart disease ketone data; KD associated with worse outcomes post-MI in some models MDPI / Koutnik et al. (2020) "Ketones Elicit Distinct Alterations in Adipose Mitochondrial Bioenergetics" International Journal of Molecular Sciences — DOI 10.3390/ijms21176255 Used for: β-hydroxybutyrate increases respiration without commensurate ATP production; elevated O2:ATP ratio confirming uncoupling; UCP1 and PGC1α upregulation Sullivan et al. / ResearchGate (2004) "The Ketogenic Diet Increases Mitochondrial Uncoupling Protein Levels and Activity" Used for: Ketogenic diet increases hippocampal uncoupling proteins; decreased ROS but at cost of coupling efficiency

    32 min
  4. Mar 24

    49. Fix Your Blood Sugar Without Cutting Carbs With Isaac Pohlman

    In this episode, Zane sits down with registered dietitian and Type 1 diabetic Isaac Pohlman to challenge one of the biggest misconceptions in modern health: that carbs are the problem. Isaac shares his powerful personal journey—from a high-level athlete dealing with chronic fatigue, hormonal issues, and gut dysfunction to being diagnosed with Type 1 diabetes in grad school. What followed wasn't just managing symptoms, but completely rethinking metabolism, energy production, and how the body actually uses fuel. Together, Zane and Isaac break down why the fear around carbohydrates is not only misguided—but often making blood sugar issues worse. They unpack the real drivers of insulin resistance, including stress, circadian rhythm disruption, ultra-processed foods, and loss of muscle mass. This conversation goes far beyond "carbs vs. no carbs." It reframes metabolic health as a system—one that includes lifestyle, light exposure, recovery, food quality, and nervous system regulation. If you've been told to cut carbs to fix your blood sugar… this episode will challenge everything you thought you kne 👇 Download Zane's Free Plan https://free40plan.com/ Follow Zane IG: https://www.instagram.com/zanegriggsfitness Guest: https://www.thepohlmaninstitute.com https://www.facebook.com/isaacpohlmannutrition https://www.instagram.com/isaacpohlman https://www.youtube.com/@isaacpohlman Where to learn more about the Pohlman Institute's work and protocols Carb Sweet Spot Masterclass [FREE] https://programs.isaacpohlman.com/offers/FoFv4San Have Your Carbs and Eat 'Em Too Book https://book.isaacpohlman.com/book-sales-page The Pohlman Method - 1:1 Coaching: https://www.thepohlmaninstitute.com/the-pohlman-method 00:00 – Intro: Why cardio & diet aren't working anymore 01:00 – Isaac's background & metabolic health approach 03:00 – Athlete to chronic fatigue: early warning signs 05:00 – Stress, trauma & the link to autoimmune conditions 07:00 – The moment metabolism changed everything 10:00 – Type 1 diabetes diagnosis & symptoms 13:00 – Why carbs are misunderstood 15:00 – Fear of carbs & blood sugar spikes explained 17:00 – Why blood sugar SHOULD rise after meals 19:00 – Insulin, thyroid & energy production connection 21:00 – The real cause of insulin resistance 23:00 – Why low-carb can backfire 26:00 – Stress hormones, muscle loss & metabolism 29:00 – How to actually reverse pre-diabetes 31:00 – Lifestyle before diet (what most people miss) 34:00 – Food density & why your meals aren't satisfying 37:00 – What's REALLY causing the diabetes epidemic 40:00 – Light exposure, circadian rhythm & blood sugar 42:00 – Ultra-processed foods & fat storage 44:00 – Personal fat threshold explained 47:00 – Getting off medication naturally 50:00 – Reintroducing carbs safely 54:00 – Best carbs for blood sugar control

    1h 1m
  5. Mar 13

    47. The Real Reason You're Always Tired After 40

    Most people think fatigue is a sleep problem. But the truth is… sleep is often just a symptom. In this episode, Zane breaks down why so many people—especially after 40—feel exhausted even when they're getting 7–8 hours of sleep. The real issue often comes from under-fueling, chronic fasting, low carb diets, and excessive training, which drive up stress hormones like cortisol and disrupt recovery. Zane shares the mistakes he made for years with fasted workouts, low-carb dieting, and overtraining, and explains how these habits can impact: • Thyroid function • Testosterone levels • Sleep quality • HRV and recovery • Energy and mood You'll also learn how to properly fuel your body for better energy, deeper sleep, and sustainable performance. If you're tired all the time, this episode could completely change how you think about recovery. Download Zane's Free Fit Over 40 Plan: 👉 https://free40plan.com 00:00 Why You're Still Tired 00:45 Sleep Is a Result of Your Day 02:10 The Hidden Cause of Poor Sleep 04:00 My Personal Low Carb & Fasting Mistake 05:40 Underfueling and Stress Hormones 07:10 Cortisol, Adrenaline, and Glucose Production 09:30 How Fasting Disrupts Sleep 11:30 Why You Wake Up at 2–3 AM 13:00 Fasted Workouts and Hormone Stress 15:10 Why Recovery Changes After 40 17:00 HRV, Heart Rate, and Recovery 18:40 The Mood and Energy Connection 20:10 Why Metabolic Stress Isn't Strength 22:00 Eat Less, Move More Is Wrong 23:40 How Much Protein You Actually Need 25:20 The Right Way to Eat Carbohydrates 27:30 Foods That Support Recovery 29:00 Training Smarter After 40 30:20 Managing Stress and Recovery 31:50 Why Sleep Starts When You Wake Up 33:10 Strategy Beats Extreme Discipline 35:20 Why Fatigue Is a Fuel Problem 36:00 Free Fit Over 40 Plan

    34 min
  6. Mar 12

    46. Low Carb Is Wrecking Your Thyroid (And Most People Don't Know It)

    Many people struggling with fatigue, weight gain, brain fog, or low libido may actually be dealing with thyroid dysfunction — and not even know it. In this episode of Healthy AF, Zane explains how common lifestyle habits like low-carb dieting, intermittent fasting, overtraining, chronic stress, and sleep deprivation can quietly disrupt thyroid function and slow your metabolism. You'll learn why markers like T3, reverse T3, and TSH matter more than most standard lab panels suggest, how the body adapts to low energy availability, and why many people with thyroid symptoms are often told their labs look "normal." Zane also breaks down the role of insulin, leptin, liver glycogen, and carbohydrate intake in thyroid hormone conversion — and why reintroducing the right foods can often restore metabolic function. If you've ever struggled with low energy, stubborn weight gain, cold intolerance, or declining performance despite dieting and training harder, this episode will help you understand what may really be happening inside your metabolism. 👇 Download Zane's Free Plan https://free40plan.com/ Follow Zane IG: https://www.instagram.com/zanegriggsfitness   Supportive research Low carb diet increases incidence of REDs and low T3⁠ https://pubmed.ncbi.nlm.nih.gov/40639795/%E2%81%A0 ⁠ Low carb diet reduces T3 in athletes⁠ https://blogs.bmj.com/bjsm/2023/08/14/a-bad-situation-made-worse-low-carbohydrate-intake-amplifies-low-energy-availability-hormonal-disturbances/⁠ ⁠ Low carb diet induces low thyroid function⁠ https://pmc.ncbi.nlm.nih.gov/articles/PMC11558958/⁠ ⁠ Keto diet reduces thyroid function⁠ https://journals.plos.org/plosone/article?id=10.1371%2Fjournal.pone.0269440⁠ www.free40plan.com   00:00 – Introduction: Thyroid Health and Metabolism 00:40 – Remembering Charlie 02:00 – Common Symptoms of Low Thyroid 03:30 – How Thyroid Affects Metabolism and Energy 04:30 – Hormonal and Cardiovascular Effects of Thyroid Dysfunction 05:30 – Why Thyroid Issues Often Go Undiagnosed 06:30 – Stress, Overtraining, and Aggressive Dieting 08:00 – How Thyroid Hormones Work (T4, T3, Reverse T3) 09:40 – What Low-Carb Diets Do to Thyroid Function 11:00 – Low Energy Availability in Athletes 12:30 – Why Your Body Interprets Low Carb as Scarcity 14:00 – The Impact of Fasting on Thyroid Hormones 15:30 – Liver Glycogen and Overnight Stress Hormones 17:00 – Why High Performers Are Vulnerable 18:30 – Insulin, Leptin, and Thyroid Signaling 20:00 – Optimal Thyroid Lab Ranges Explained 22:30 – Why Symptoms Matter More Than "Normal" Lab Ranges 24:30 – How Thyroid Issues Affect Testosterone in Men 26:00 – Insulin Signaling and Thyroid Conversion 28:00 – Low Carb vs Insulin Resistance: Different Labs, Same Symptoms 31:00 – Why Leptin Is the Missing Link 33:00 – Reintroducing Carbs to Restore Thyroid Function 35:00 – LDL Changes After Increasing Carbohydrates 36:30 – Body Temperature and Metabolic Rate 37:30 – Training, Recovery, and Sleep 38:30 – Best Carbohydrate Sources for Thyroid Health 41:00 – How Much Carbohydrate You Actually Need 43:00 – Macro Ratios for Fat Loss vs Performance 45:00 – What Indigenous Diets Teach Us About Metabolic Health 47:00 – How Ultra-Processed Foods Disrupt Energy Production 49:00 – The Real Cause of Fat Gain and Metabolic Dysfunction 51:00 – Why "Calories In Calories Out" Misses the Real Problem 53:00 – Fixing Metabolism Through Food Quality and Energy Availability

    57 min
  7. Mar 10

    45. Most Men Over 40 Are Missing These 5 Blood Markers

    Most men focus on body weight, workouts, or body fat… but those numbers don't tell the full story. In this episode of Healthy AF, Zane breaks down the five blood markers every man over 40 should be tracking if they want to stay strong, energized, and metabolically healthy for decades to come. These biomarkers can reveal problems years before symptoms appear and help you catch issues related to metabolic dysfunction, heart disease, inflammation, testosterone decline, and prostate health early. You'll learn why markers like fasting insulin and fasting glucose often reveal metabolic issues long before diabetes develops, how free testosterone reflects metabolic stress, and why inflammation markers like hs-CRP matter for long-term health. Zane also explains how lifestyle factors such as poor sleep, chronic stress, excessive fasting, overtraining, and ultra-processed foods can quietly push these numbers in the wrong direction. If you're over 40 and serious about longevity, performance, and metabolic health, these are the numbers you need to know. 👇 Download Zane's Free Plan https://free40plan.com/ Follow Zane IG: https://www.instagram.com/zanegriggsfitness/ 00:00 – 5 Blood Markers Every Man Over 40 Should Know 00:55 – Why Body Weight and Workouts Aren't Enough 01:40 – Marker #1: Fasting Insulin (Earliest Warning Sign) 02:50 – Ideal Fasting Insulin Range 04:00 – What Causes Insulin Resistance 05:05 – Marker #2: Fasting Glucose 06:00 – Why Chronically High Glucose Damages Blood Vessels 07:20 – Low-Carb, Fasting, and Elevated Blood Sugar 08:25 – Ideal Blood Sugar Range 09:15 – Stress Hormones and the "Dawn Effect" 11:00 – Marker #3: Free Testosterone 12:00 – Stress, Fasting, and Testosterone Suppression 13:20 – Ideal Free Testosterone Range 14:10 – Marker #4: hs-CRP (Inflammation Marker) 15:00 – What Drives Chronic Inflammation 15:40 – Visceral Fat, Diet, and Alcohol Effects 16:00 – Marker #5: PSA (Prostate Health) 17:00 – Testosterone and Prostate Cancer Myths 18:20 – Why You Should Start Tracking These Markers Early 19:10 – Why "Normal" Lab Ranges Aren't Optimal

    22 min
5
out of 5
12 Ratings

About

Healthy AF, the podcast dedicated to helping you live a healthier life after the age of 50, hosted by health and longevity expert Zane Griggs. This show is your guide to achieving optimal health, both physically and mentally. Whether you're in your 20s and want to plan for a healthy future or in your 50s and seeking to catch up, this show could help you improve your nutrition, fitness, and overall well-being. Each week, we'll dive into the latest research and expert advice on topics such as exercise, stress management, and more. We'll discuss practical strategies for maintaining a healthy lifestyle, including how to balance your diet and stay active as you age. Remember, it's never too late to prioritize your health and become Healthy AF! (Hit play to learn more)

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